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264 Section II / General Surgery

TYPES OF SURGERIES

Define the following terms:

Graham patch For treatment of duodenal perforation in poor operative candidates/unstable patients

Place viable omentum over perforation and tack into place with sutures

Truncal vagotomy

Resection of a 1- to 2-cm segment of

 

each vagal trunk as it enters the

 

abdomen on the distal esophagus,

 

decreasing gastric acid secretion

What other procedure must be performed along with a truncal vagotomy?

“Drainage procedure” (pyloroplasty, antrectomy, or gastrojejunostomy), because vagal fibers provide relaxation of the pylorus, and, if you cut them, the pylorus will not open

Define the following terms:

Vagotomy and pyloroplasty Pyloroplasty performed with vagotomy to compensate for decreased gastric emptying

Chapter 40 / Upper GI Bleeding 265

Vagotomy and antrectomy Remove antrum and pylorus in addition to vagotomy; reconstruct as a Billroth I or II

What is the goal of duodenal Decrease gastric acid secretion (and fix ulcer surgery? IHOP)

What is the advantage of No drainage procedure is needed; vagal proximal gastric vagotomy fibers to the pylorus are preserved; rate (highly selective of dumping syndrome is low vagotomy)?

What is a Billroth I (BI)?

Truncal vagotomy, antrectomy, and gastroduodenostomy (Think: BI ONE limb off of the stomach remnant)

What are the contraindica-

Gastric cancer or suspicion of gastric

tions for a Billroth I?

cancer

266 Section II / General Surgery

 

What is a Billroth II (BII)?

Truncal vagotomy, antrectomy, and

 

gastrojejunostomy (Think: BII TWO

 

limbs off of the stomach remnant)

What is the Kocher

Dissect the left lateral peritoneal

maneuver?

attachments to the duodenum to allow

 

visualization of posterior duodenum

STRESS GASTRITIS

 

 

 

What is it?

Superficial mucosal erosions in the

 

stressed patient

What are the risk factors?

Sepsis, intubation, trauma, shock, burn,

 

brain injury

What is the prophylactic

H2 blockers, PPIs, antacids, sucralfate

treatment?

 

What are the signs/symptoms?

NGT blood (usually), painless (usually)

How is it diagnosed?

EGD, if bleeding is significant

What is the treatment for

LAVAGE out blood clots, give a maximum

gastritis?

dose of PPI in a 24-hour IV drip

MALLORY-WEISS SYNDROME

 

 

What is it?

Post-retching, postemesis longitudinal

 

tear (submucosa and mucosa) of the

 

stomach near the GE junction; approxi-

 

mately three fourths are in the stomach

Chapter 40 / Upper GI Bleeding 267

For what percentage of all

10%

upper GI bleeds does this syndrome account?

What are the causes of a tear?

What are the risk factors?

What are the symptoms?

What percentage of patients will have hematemesis?

How is the diagnosis made?

What is the “classic” history?

Increased gastric pressure, often aggravated by hiatal hernia

Retching, alcoholism (50%), 50% of patients have hiatal hernia

Epigastric pain, thoracic substernal pain, emesis, hematemesis

85%

EGD

Alcoholic patient after binge drinking— first, vomit food and gastric contents, followed by forceful retching and bloody vomitus

What is the treatment?

When is surgery indicated?

Can the SengstakenBlakemore tamponade balloon be used for treatment of Mallory-Weiss tear bleeding?

Room temperature water lavage (90% of patients stop bleeding), electrocautery, arterial embolization, or surgery for refractory bleeding

When medical/endoscopic treatment fails ( 6 u PRBCs infused)

No, it makes bleeding worse

Use the balloon only for bleeding from esophageal varices

ESOPHAGEAL VARICEAL BLEEDING

What is it?

Bleeding from formation of esophageal

 

varices from back up of portal pressure

 

via the coronary vein to the submucosal

 

esophageal venous plexuses secondary to

 

portal hypertension from liver cirrhosis

268 Section II / General Surgery

What is the “rule of two thirds” of esophageal variceal hemorrhage?

What are the signs/ symptoms?

How is the diagnosis made?

What is the acute medical treatment?

In the patient with CAD, what must you give in addition to the vasopressin?

What are the treatment options?

What is the SengstakenBlakemore balloon?

Two thirds of patients with portal hypertension develop esophageal varices

Two thirds of patients with esophageal varices bleed

Liver disease, portal hypertension, hematemesis, caput medusa, ascites

EGD (very important because only 50% of UGI bleeding in patients with known esophageal varices are bleeding from the varices; the other 50% have bleeding from ulcers, etc.)

Lower portal pressure with somatostatin and vasopressin

Nitroglycerin—to prevent coronary artery vasoconstriction that may result in an MI

Sclerotherapy or band ligation via endoscope, TIPS, liver transplant

Tamponades with an esophageal balloon and a gastric balloon

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