- •Preface
- •List of contributers
- •History, epidemiology, prevention and education
- •A history of burn care
- •“Black sheep in surgical wards”
- •Toxaemia, plasmarrhea, or infection?
- •The Guinea Pig Club
- •Burns and sulfa drugs at Pearl Harbor
- •Burn center concept
- •Shock and resuscitation
- •Wound care and infection
- •Burn surgery
- •Inhalation injury and pulmonary care
- •Nutrition and the “Universal Trauma Model”
- •Rehabilitation
- •Conclusions
- •References
- •Epidemiology and prevention of burns throughout the world
- •Introduction
- •Epidemiology
- •The inequitable distribution of burns
- •Cost by age
- •Cost by mechanism
- •Limitations of data
- •Risk factors
- •Socioeconomic factors
- •Race and ethnicity
- •Age-related factors: children
- •Age-related factors: the elderly
- •Regional factors
- •Gender-related factors
- •Intent
- •Comorbidity
- •Agents
- •Non-electric domestic appliances
- •War, mass casualties, and terrorism
- •Interventions
- •Smoke detectors
- •Residential sprinklers
- •Hot water temperature regulation
- •Lamps and stoves
- •Fireworks legislation
- •Fire-safe cigarettes
- •Children’s sleepwear
- •Acid assaults
- •Burn care systems
- •Role of the World Health Organization
- •Conclusions and recommendations
- •Surveillance
- •Smoke alarms
- •Gender inequality
- •Community surveys
- •Acknowledgements
- •References
- •Prevention of burn injuries
- •Introduction
- •Burns prevalence and relevance
- •Burn injury risk factors
- •WHERE?
- •Burn prevention types
- •Burn prevention: The basics to design a plan
- •Flame burns
- •Prevention of scald burns
- •Conclusions
- •References
- •Burns associated with wars and disasters
- •Introduction
- •Wartime burns
- •Epidemiology of burns sustained during combat operations
- •Fluid resuscitation and initial burn care in theater
- •Evacuation of thermally-injured combat casualties
- •Care of host-nation burn patients
- •Disaster-related burns
- •Epidemiology
- •Treatment of disaster-related burns
- •The American Burn Association (ABA) disaster management plan
- •Summary
- •References
- •Education in burns
- •Introduction
- •Surgical education
- •Background
- •Simulation
- •Education in the internet era
- •Rotations as courses
- •Mentorship
- •Peer mentorship
- •Hierarchical mentorship
- •What is a mentor
- •Implementation
- •Interprofessional education
- •What is interprofessional education
- •Approaches to interprofessional education
- •References
- •European practice guidelines for burn care: Minimum level of burn care provision in Europe
- •Foreword
- •Background
- •Introduction
- •Burn injury and burn care in general
- •Conclusion
- •References
- •Pre-hospital and initial management of burns
- •Introduction
- •Modern care
- •Early management
- •At the accident
- •At a local hospital – stabilization prior to transport to the Burn Center
- •Transportation
- •References
- •Medical documentation of burn injuries
- •Introduction
- •Medical documentation of burn injuries
- •Contents of an up-to-date burns registry
- •Shortcomings in existing documentation systems designs
- •Burn depth
- •Burn depth as a dynamic process
- •Non-clinical methods to classify burn depth
- •Burn extent
- •Basic principles of determining the burn extent
- •Methods to determine burn extent
- •Computer aided three-dimensional documentation systems
- •Methods used by BurnCase 3D
- •Creating a comparable international database
- •Results
- •Conclusion
- •Financing and accomplishment
- •References
- •Pathophysiology of burn injury
- •Introduction
- •Local changes
- •Burn depth
- •Burn size
- •Systemic changes
- •Hypovolemia and rapid edema formation
- •Altered cellular membranes and cellular edema
- •Mediators of burn injury
- •Hemodynamic consequences of acute burns
- •Hypermetabolic response to burn injury
- •Glucose metabolism
- •Myocardial dysfunction
- •Effects on the renal system
- •Effects on the gastrointestinal system
- •Effects on the immune system
- •Summary and conclusion
- •References
- •Anesthesia for patients with acute burn injuries
- •Introduction
- •Preoperative evaluation
- •Monitors
- •Pharmacology
- •Postoperative care
- •References
- •Diagnosis and management of inhalation injury
- •Introduction
- •Effects of inhaled gases
- •Carbon monoxide
- •Cyanide toxicity
- •Upper airway injury
- •Lower airway injury
- •Diagnosis
- •Resuscitation after inhalation injury
- •Other treatment issues
- •Prognosis
- •Conclusions
- •References
- •Respiratory management
- •Airway management
- •(a) Endotracheal intubation
- •(b) Elective tracheostomy
- •Chest escharotomy
- •Conventional mechanical ventilation
- •Introduction
- •Pathophysiological principles
- •Low tidal volume and limited plateau pressure approaches
- •Permissive hypercapnia
- •The open-lung approach
- •PEEP
- •Lung recruitment maneuvers
- •Unconventional mechanical ventilation strategies
- •High-frequency percussive ventilation (HFPV)
- •High-frequency oscillatory ventilation
- •Airway pressure release ventilation (APRV)
- •Ventilator associated pneumonia (VAP)
- •(a) Prevention
- •(b) Treatment
- •References
- •Organ responses and organ support
- •Introduction
- •Burn shock and resuscitation
- •Post-burn hypermetabolism
- •Individual organ systems
- •Central nervous system
- •Peripheral nervous system
- •Pulmonary
- •Cardiovascular
- •Renal
- •Gastrointestinal tract
- •Conclusion
- •References
- •Critical care of thermally injured patient
- •Introduction
- •Oxidative stress control strategies
- •Fluid and cardiovascular management beyond 24 hours
- •Other organ function/dysfunction and support
- •The nervous system
- •Respiratory system and inhalation injury
- •Renal failure and renal replacement therapy
- •Gastro-intestinal system
- •Glucose control
- •Endocrine changes
- •Stress response (Fig. 2)
- •Low T3 syndrome
- •Gonadal depression
- •Thermal regulation
- •Metabolic modulation
- •Propranolol
- •Oxandrolone
- •Recombinant human growth hormone
- •Insulin
- •Electrolyte disorders
- •Sodium
- •Chloride
- •Calcium, phosphate and magnesium
- •Calcium
- •Bone demineralization and osteoporosis
- •Micronutrients and antioxidants
- •Thrombosis prophylaxis
- •Conclusion
- •References
- •Treatment of infection in burns
- •Introduction
- •Clinical management strategies
- •Pathophysiology of the burn wound
- •Burn wound infection
- •Cellulitis
- •Impetigo
- •Catheter related infections
- •Urinary tract infection
- •Tracheobronchitis
- •Pneumonia
- •Sepsis in the burn patient
- •The microbiology of burn wound infection
- •Sources of organisms
- •Gram-positive organisms
- •Gram-negative organisms
- •Infection control
- •Pharmacological considerations in the treatment of burn infections
- •Topical antimicrobial treatment
- •Systemic antimicrobial treatment (Table 3)
- •Gram-positive bacterial infections
- •Enterococcal bacterial infections
- •Gram-negative bacterial infections
- •Treatment of yeast and fungal infections
- •The Polyenes (Amphotericin B)
- •Azole antifungals
- •Echinocandin antifungals
- •Nucleoside analog antifungal (Flucytosine)
- •Conclusion
- •References
- •Acute treatment of severely burned pediatric patients
- •Introduction
- •Initial management of the burned child
- •Fluid resuscitation
- •Sepsis
- •Inhalation injury
- •Burn wound excision
- •Burn wound coverage
- •Metabolic response and nutritional support
- •Modulation of the hormonal and endocrine response
- •Recombinant human growth hormone
- •Insulin-like growth factor
- •Oxandrolone
- •Propranolol
- •Glucose control
- •Insulin
- •Metformin
- •Novel therapeutic options
- •Long-term responses
- •Conclusion
- •References
- •Adult burn management
- •Introduction
- •Epidemiology and aetiology
- •Pathophysiology
- •Assessment of the burn wound
- •Depth of burn
- •Size of the burn
- •Initial management of the burn wound
- •First aid
- •Burn blisters
- •Escharotomy
- •General care of the adult burn patient
- •Biological/Semi biological dressings
- •Topical antimicrobials
- •Biological dressings
- •Other dressings
- •Exposure
- •Deep partial thickness wound
- •Total wound excision
- •Serial wound excision and conservative management
- •Full thickness burns
- •Excision and autografting
- •Topical antimicrobials
- •Large full thickness burns
- •Serial excision
- •Mixed depth burn
- •Donor sites
- •Techniques of wound excision
- •Blood loss
- •Antibiotics
- •Anatomical considerations
- •Skin replacement
- •Autograft
- •Allograft
- •Other skin replacements
- •Cultured skin substitutes
- •Skin graft take
- •Rehabilitation and outcome
- •Future care
- •References
- •Burns in older adults
- •Introduction
- •Burn injury epidemiology
- •Pathophysiologic changes and implications for burn therapy
- •Aging
- •Comorbidities
- •Acute management challenges
- •Fluid resuscitation
- •Burn excision
- •Pain and sedation
- •End of life decisions
- •Summary of key points and recommendations
- •References
- •Acute management of facial burns
- •Introduction
- •Anatomy and pathophysiology
- •Management
- •General approach
- •Airway management
- •Facial burn wound management
- •Initial wound care
- •Topical agents
- •Biological dressings
- •Surgical burn wound excision of the face
- •Wound closure
- •Special areas and adjacent of the face
- •Eyelids
- •Nose and ears
- •Lips
- •Scalp
- •The neck
- •Catastrophic injury
- •Post healing rehabilitation and scar management
- •Outcome and reconstruction
- •Summary
- •References
- •Hand burns
- •Introduction
- •Initial evaluation and history
- •Initial wound management
- •Escharotomy and fasciotomy
- •Surgical management: Early excision and grafting
- •Skin substitutes
- •Amputation
- •Hand therapy
- •Secondary reconstruction
- •References
- •Treatment of burns – established and novel technology
- •Introduction
- •Partial thickness burns
- •Biological membranes – amnion and others
- •Xenograft
- •Full thickness burns
- •Dermal analogs
- •Keratinocyte coverage
- •Facial transplantation
- •Tissue engineering and stem cells
- •Gene therapy and growth factors
- •Conclusion
- •References
- •Wound healing
- •History of wound care
- •Types of wounds
- •Mechanisms of wound healing
- •Hemostasis
- •Proliferation
- •Epithelialization
- •Remodeling
- •Fetal wound healing
- •Stem cells
- •Abnormal wound healing
- •Impaired wound healing
- •Hypertrophic scars and keloids
- •Chronic non-healing wounds
- •Conclusions
- •References
- •Pain management after burn trauma
- •Introduction
- •Pathophysiology of pain after burn injuries
- •Nociceptive pain
- •Neuropathic pain
- •Sympathetically Maintained Pain (SMP)
- •Pain rating and documentation
- •Pain management and analgesics
- •Pharmacokinetics in severe burns
- •Form of administration [21]
- •Non-opioids (Table 1)
- •Paracetamol
- •Metamizole
- •Non-steroidal antirheumatics (NSAID)
- •Selective cyclooxygenasis-2-inhibitors
- •Opioids (Table 2)
- •Weak opioids
- •Strong opioids
- •Other analgesics
- •Ketamine (see also intensive care unit and analgosedation)
- •Anticonvulsants (Gabapentin and Pregabalin)
- •Antidepressants with analgesic effects
- •Regional anesthesia
- •Pain management without analgesics
- •Adequate communication
- •Psychological techniques [65]
- •Transcutaneous electrical nerve stimulation (TENS)
- •Particularities of burn pain
- •Wound pain
- •Breakthrough pain
- •Intervention-induced pain
- •Necrosectomy and skin grafting
- •Dressing change of large burn wounds and removal of clamps in skin grafts
- •Dressing change in smaller burn wounds, baths and physical therapy
- •Postoperative pain
- •Mental aspects
- •Intensive care unit
- •Opioid-induced hyperalgesia and opioid tolerance
- •Hypermetabolism
- •Psychic stress factors
- •Risk of infection
- •Monitoring [92]
- •Sedation monitoring
- •Analgesia monitoring (see Fig. 2)
- •Analgosedation (Table 3)
- •Sedation
- •Analgesia
- •References
- •Nutrition support for the burn patient
- •Background
- •Case presentation
- •Patient selection: Timing and route of nutritional support
- •Determining nutritional demands
- •What is an appropriate initial nutrition plan for this patient?
- •Formulations for nutritional support
- •Monitoring nutrition support
- •Optimal monitoring of nutritional status
- •Problems and complications of nutritional support
- •Conclusion
- •References
- •HBO and burns
- •Historical development
- •Contraindications for the use of HBO
- •Conclusion
- •References
- •Nursing management of the burn-injured person
- •Introduction
- •Incidence
- •Prevention
- •Pathophysiology
- •Severity factors
- •Local damage
- •Fluid and electrolyte shifts
- •Cardiovascular, gastrointestinal and renal system manifestations
- •Types of burn injuries
- •Thermal
- •Chemical
- •Electrical
- •Smoke and inhalation injury
- •Clinical manifestations
- •Subjective symptoms
- •Possible complications
- •Clinical management
- •Non-surgical care
- •Surgical care
- •Coordination of care: Burn nursing’s unique role
- •Nursing interventions: Emergent phase
- •Nursing interventions: Acute phase
- •Nursing interventions: Rehabilitative phase
- •Ongoing care
- •Infection prevention and control
- •Rehabilitation medicine
- •Nutrition
- •Pharmacology
- •Conclusion
- •References
- •Outpatient burn care
- •Introduction
- •Epidemiology
- •Accident causes
- •Care structures
- •Indications for inpatient treatment
- •Patient age
- •Total burned body surface area (TBSA)
- •Depth of the burn
- •Pre-existing conditions
- •Accompanying injuries
- •Special injuries
- •Treatment
- •Initial treatment
- •Pain therapy
- •Local treatment
- •Course of treatment
- •Complications
- •Infections
- •Follow-up care
- •References
- •Non-thermal burns
- •Electrical injury
- •Introduction
- •Pathophysiology
- •Initial assessment and acute care
- •Wound care
- •Diagnosis
- •Low voltage injuries
- •Lightning injuries
- •Complications
- •References
- •Symptoms, diagnosis and treatment of chemical burns
- •Chemical burns
- •Decontamination
- •Affection of different organ systems
- •Respiratory tract
- •Gastrointestinal tract
- •Hematological signs
- •Nephrologic symptoms
- •Skin
- •Nitric acid
- •Sulfuric acid
- •Caustic soda
- •Phenol
- •Summary
- •References
- •Necrotizing and exfoliative diseases of the skin
- •Introduction
- •Necrotizing diseases of the skin
- •Cellulitis
- •Staphylococcal scalded skin syndrome
- •Autoimmune blistering diseases
- •Epidermolysis bullosa acquisita
- •Necrotizing fasciitis
- •Purpura fulminans
- •Exfoliative diseases of the skin
- •Stevens-Johnson syndrome
- •Toxic epidermal necrolysis
- •Conclusion
- •References
- •Frostbite
- •Mechanism
- •Risk factors
- •Causes
- •Diagnosis
- •Treatment
- •Rewarming
- •Surgery
- •Sympathectomy
- •Vasodilators
- •Escharotomy and fasciotomy
- •Prognosis
- •Research
- •References
- •Subject index
F. Sjöberg
plicate the management, prolong recovery or effect mortality
Hospitals without qualified personal or equipment for the care of the critically burned children
Transportation
Transportation of the burn victim may involve several steps, – but most often two. The first, is from the site of the accident to a local hospital, or to a similar point for stabilization. The second transport is from the referring hospital to the burn center, where the final treatment is provided. The first transport distance is often short and need for planning is less. Most often in Europe this is done by ambulance and the care during this transport is provided by paramedics or nurses which are stationed in the ambulance. The activities that have been undertaken at the scene of the accidents and during transport are then reported by the paramedics/nurse and/or documented in their report, which may be of complementary value when receiving the patient at the local hospital. In the report data regarding the patient, the circumstances at the scene as well as surveillance data may be found. At this point it is also important to identify the patient and obtain relevant data regarding relatives so that information can be passed on or complementary questions regarding the background of the patient can be obtained.
The second transport is most often done from a local hospital, where the patient has been stabilized and some important burn related treatments have been commenced, such as: intubation/ventilatory treatment in cases of compromised airway; fluid treatment for burn shock. In cases of circulatory compromise escharotomies should have been performed. Important is also that in the early care other trauma induced injuries should have been diagnosed and attended to, – especially if urgent and/or life threatening.
The choice of transport means depend on several factors of which local geography may be important, e. g., in an island or in an archipelago where airborne transport is almost obligatory. In general, transport exceeding 100 km often calls for airborne transport, such as helicopters or aircraft. Smaller hospitals may not have a helicopter landing facility
and the first transport then involves an ambulance transport to the airfield. Tertiary referral hospitals (burn centers) in Europe most often have helicopter landing facilities. Specifically, if the patient needs ventilator or other specific intensive care treatments or interventions during transport a specially designed intensive care type ambulance is needed (Figs. 5 and 6).
It is important to stress the need for monitoring during transport, especially in major burns and in ventilated patients. For these patients active heating devices, ventilators and invasive blood pressure monitoring is relevant. It is important for the referring physician to be aware of the monitoring facilities provided by each type of transport system as this may pose a risk if the patient is not properly monitored and/or if interventions if needed are difficult or impossible to undertake during the transport. In some smaller helicopter types critical care interventions may at times be difficult to perform and for such situations ground transport may be preferred. Also the referring physician needs to be updated on the skills and training of the transport surveillance personal, who should be properly trained and have the relevant equipment for the transport that is planned.
Fig. 5. Transportable critical care bed including equipment. Mobile critical care bed, developed for the aircraft emergency services (Sweden). It contains ventilator, breathing gas supplies, intravenous infusion systems as well as monitoring devices. The equipment is standardized so it facilitates transports involving both vehicles and aircraft in the same transport procedure (with permission Liber AB)
114
Pre-hospital, fluid and early management, burn wound evaluation
Fig. 6. Large interior of ambulance prepared for transporting critically ill patients. The space aside the bed facilitates critical care procedures during transport. It is also feasible to embark a standard critical care bed for transport purposes. The cabin holds complete power supplies as well as mounting racks for standard ventilators, infusion pumps as well as for patient surveillance equipment (with permission Liber AB)
References
[1]Akerlund E, Huss FR, Sjoberg F (2007) Burns in Sweden: an analysis of 24,538 cases during the period 1987–2004. Burns 33(1): 31–36
[2]Brigham PA, McLoughlin E (1996) Burn incidence and medical care use in the United States: estimates, trends, and data sources. J Burn Care Rehabil 17(2): 95–107
[3]Andel D, Kamolz LP, Niedermayr M, Hoerauf K, Schramm W, Andel H (2007) Which of the abbreviated burn severity index variables are having impact on the hospital length of stay? J Burn Care Res 28(1): 163–166
[4]Still JM, Jr, Law EJ, Belcher K, Thiruvaiyarv D (1996) Decreasing length of hospital stay by early excision and grafting of burns. South Med J 89(6): 578–582
[5] Gomez M, Cartotto R, Knighton J, Smith K, Fish JS (2008) Improved survival following thermal injury in adult patients treated at a regional burn center. J Burn Care Res 29(1): 130–137
[6]Miller SF, Bessey PQ, Schurr MJ, Browning SM, Jeng JC, Caruso DM et al (2006) National burn repository 2005: a ten-year review. J Burn Care Res 27(4): 411–436
[7]Mertens DM, Jenkins ME, Warden GD (1997) Outpatient burn management. Nurs Clin North Am 32(2): 343–364
[8]Moss LS (2004) Outpatient management of the burn patient. Crit Care Nurs Clin North Am 16(1): 109–117
[9]Tompkins D, Rossi LA (2004) Care of out patient burns. Burns 30(8):A7–9
[10]Bessey PQ, Arons RR, Dimaggio CJ, Yurt RW (2006) The vulnerabilities of age: burns in children and older adults. Surgery 140(4): 705–15; discussion 15–17
[11]Sheridan R (2007) Burns at the extremes of age. J Burn Care Res 28(4): 580–585
[12]Thombs BD, Singh VA, Halonen J, Diallo A, Milner SM (2007) The effects of preexisting medical comorbidities on mortality and length of hospital stay in acute burn injury: evidence from a national sample of 31,338 adult patients. Ann Surg 245(4): 629–634
[13]Praiss IL, Feller I, James MH (1980) The planning and organization of a regionalized burn care system. Med Care 18(2): 202–210
[14]Yurt RW, Bessey PQ (2009) The development of a regional system for care of the burn-injured patients. Surg Infect (Larchmt) 10(5): 441–445
[15]Vercruysse GA, Ingram WL, Feliciano DV (2011) The demographics of modern burn care: should most burns be cared for by non-burn surgeons? Am J Surg 201(1): 91–96
[16]Munzberg M, Mahlke L, Bouillon B, Paffrath T, Matthes G, Wolfl CG (2010) [Six years of Advanced Trauma Life Support (ATLS) in Germany: The 100th provider course in Hamburg.]. Unfallchirurg 113(7): 561–566
[17]Soreide K. Three decades (1978–2008) of Advanced Trauma Life Support (ATLS) practice revised and evidence revisited. Scand J Trauma Resusc Emerg Med 16(1): 19
[18]Sasaki J, Takuma K, Oda J, Saitoh D, Takeda T, Tanaka H et al (2010) Experiences in organizing Advanced Burn Life Support (ABLS) provider courses in Japan. Burns 36(1):65–69
[19]Cochran A, Edelman LS, Morris SE, Saffle JR (2008) Learner satisfaction with Web-based learning as an adjunct to clinical experience in burn surgery. J Burn Care Res 29(1): 222–226
[20]Lindford AJ, Lamyman MJ, Lim P (2006) Review of the emergency management of severe burns (EMSB) course. Burns 32(3): 391
[21]Stone CA, Pape SA (1999) Evolution of the Emergency Management of Severe Burns (EMSB) course in the UK. Burns 25(3): 262–264
[22]Haberal M (2006) Guidelines for dealing with disasters involving large numbers of extensive burns. Burns 32(8): 933–939
[23]Cartotto R (2009) Fluid resuscitation of the thermally injured patient. Clin Plast Surg 36(4): 569–581
115
F. Sjöberg
[24]Tricklebank S (2009) Modern trends in fluid therapy for burns. Burns 35(6): 757–767
[25]Lund T, Onarheim H, Reed RK (1992) Pathogenesis of edema formation in burn injuries. World J Surg 16(1): 2–9
[26]Vlachou E, Gosling P, Moiemen NS (2006) Microalbuminuria: a marker of endothelial dysfunction in thermal injury. Burns 32(8): 1009–1016
[27]Vlachou E, Gosling P, Moiemen NS (2008) Microalbuminuria: a marker of systemic endothelial dysfunction during burn excision. Burns 34(2): 241–246
[28]Steinvall I, Bak Z, Sjoberg F (2008) Acute respiratory distress syndrome is as important as inhalation injury for the development of respiratory dysfunction in major burns. Burns 34(4): 441–451
[29]Lawrence A, Faraklas I, Watkins H, Allen A, Cochran A, Morris S et al (2010) Colloid administration normalizes resuscitation ratio and ameliorates “fluid creep”. J Burn Care Res 31(1): 40–47
[30]Saffle JI (2007) The phenomenon of “fluid creep” in acute burn resuscitation. J Burn Care Res 28(3): 382–95
[31]Baxter CR, Shires T (1968) Physiological response to crystalloid resuscitation of severe burns. Ann N Y Acad Sci 150(3): 874–894
inal compartment syndrome in patients with major burns. Burns 32(2): 151–154
[34]Holm C, Mayr M, Tegeler J, Horbrand F, Henckel von Donnersmarck G, Muhlbauer W et al (2004) A clinical randomized study on the effects of invasive monitoring on burn shock resuscitation. Burns 30(8): 798–807
[35]Bak Z, Sjoberg F, Eriksson O, Steinvall I, Janerot-Sjoberg B (2009) Hemodynamic changes during resuscitation after burns using the Parkland formula. J Trauma 66(2): 329–336
[36]Sjoberg F (2008) The ‘Parkland protocol’ for early fluid resuscitation of burns: too little, too much, or . . . even . . .
too late . . .? Acta Anaesthesiol Scand 52(6): 725–726
[37]manual. ABA ABLs. Chicago IL, 2005
[38]Choiniere M, Melzack R, Rondeau J, Girard N, Paquin MJ (1989) The pain of burns: characteristics and correlates. J Trauma 29(11): 1531–1539
[39]Raff T, Germann G, Hartmann B (1997) The value of early enteral nutrition in the prophylaxis of stress ulceration in the severely burned patient. Burns 23(4): 313–318
Correspondence: Folke Sjöberg, M. D., Ph. D., Professor, Consultant, Director, the Burn Center, Department of Hand
[32]Warden GD (1992) Burn shock resuscitation. World J and Plastic Surgery and Intensive Care, Linköping University
Surg 16(1): 16–23
[33]Oda J, Yamashita K, Inoue T, Harunari N, Ode Y, Mega K et al (2006) Resuscitation fluid volume and abdom-
Hospital, and Dept. of Clinical and Experimental Medicine, Linköping University, 581 85 Linköping, Sweden, E-mail: folke.sjoberg@liu.se
116