- •Preface
- •List of contributers
- •History, epidemiology, prevention and education
- •A history of burn care
- •“Black sheep in surgical wards”
- •Toxaemia, plasmarrhea, or infection?
- •The Guinea Pig Club
- •Burns and sulfa drugs at Pearl Harbor
- •Burn center concept
- •Shock and resuscitation
- •Wound care and infection
- •Burn surgery
- •Inhalation injury and pulmonary care
- •Nutrition and the “Universal Trauma Model”
- •Rehabilitation
- •Conclusions
- •References
- •Epidemiology and prevention of burns throughout the world
- •Introduction
- •Epidemiology
- •The inequitable distribution of burns
- •Cost by age
- •Cost by mechanism
- •Limitations of data
- •Risk factors
- •Socioeconomic factors
- •Race and ethnicity
- •Age-related factors: children
- •Age-related factors: the elderly
- •Regional factors
- •Gender-related factors
- •Intent
- •Comorbidity
- •Agents
- •Non-electric domestic appliances
- •War, mass casualties, and terrorism
- •Interventions
- •Smoke detectors
- •Residential sprinklers
- •Hot water temperature regulation
- •Lamps and stoves
- •Fireworks legislation
- •Fire-safe cigarettes
- •Children’s sleepwear
- •Acid assaults
- •Burn care systems
- •Role of the World Health Organization
- •Conclusions and recommendations
- •Surveillance
- •Smoke alarms
- •Gender inequality
- •Community surveys
- •Acknowledgements
- •References
- •Prevention of burn injuries
- •Introduction
- •Burns prevalence and relevance
- •Burn injury risk factors
- •WHERE?
- •Burn prevention types
- •Burn prevention: The basics to design a plan
- •Flame burns
- •Prevention of scald burns
- •Conclusions
- •References
- •Burns associated with wars and disasters
- •Introduction
- •Wartime burns
- •Epidemiology of burns sustained during combat operations
- •Fluid resuscitation and initial burn care in theater
- •Evacuation of thermally-injured combat casualties
- •Care of host-nation burn patients
- •Disaster-related burns
- •Epidemiology
- •Treatment of disaster-related burns
- •The American Burn Association (ABA) disaster management plan
- •Summary
- •References
- •Education in burns
- •Introduction
- •Surgical education
- •Background
- •Simulation
- •Education in the internet era
- •Rotations as courses
- •Mentorship
- •Peer mentorship
- •Hierarchical mentorship
- •What is a mentor
- •Implementation
- •Interprofessional education
- •What is interprofessional education
- •Approaches to interprofessional education
- •References
- •European practice guidelines for burn care: Minimum level of burn care provision in Europe
- •Foreword
- •Background
- •Introduction
- •Burn injury and burn care in general
- •Conclusion
- •References
- •Pre-hospital and initial management of burns
- •Introduction
- •Modern care
- •Early management
- •At the accident
- •At a local hospital – stabilization prior to transport to the Burn Center
- •Transportation
- •References
- •Medical documentation of burn injuries
- •Introduction
- •Medical documentation of burn injuries
- •Contents of an up-to-date burns registry
- •Shortcomings in existing documentation systems designs
- •Burn depth
- •Burn depth as a dynamic process
- •Non-clinical methods to classify burn depth
- •Burn extent
- •Basic principles of determining the burn extent
- •Methods to determine burn extent
- •Computer aided three-dimensional documentation systems
- •Methods used by BurnCase 3D
- •Creating a comparable international database
- •Results
- •Conclusion
- •Financing and accomplishment
- •References
- •Pathophysiology of burn injury
- •Introduction
- •Local changes
- •Burn depth
- •Burn size
- •Systemic changes
- •Hypovolemia and rapid edema formation
- •Altered cellular membranes and cellular edema
- •Mediators of burn injury
- •Hemodynamic consequences of acute burns
- •Hypermetabolic response to burn injury
- •Glucose metabolism
- •Myocardial dysfunction
- •Effects on the renal system
- •Effects on the gastrointestinal system
- •Effects on the immune system
- •Summary and conclusion
- •References
- •Anesthesia for patients with acute burn injuries
- •Introduction
- •Preoperative evaluation
- •Monitors
- •Pharmacology
- •Postoperative care
- •References
- •Diagnosis and management of inhalation injury
- •Introduction
- •Effects of inhaled gases
- •Carbon monoxide
- •Cyanide toxicity
- •Upper airway injury
- •Lower airway injury
- •Diagnosis
- •Resuscitation after inhalation injury
- •Other treatment issues
- •Prognosis
- •Conclusions
- •References
- •Respiratory management
- •Airway management
- •(a) Endotracheal intubation
- •(b) Elective tracheostomy
- •Chest escharotomy
- •Conventional mechanical ventilation
- •Introduction
- •Pathophysiological principles
- •Low tidal volume and limited plateau pressure approaches
- •Permissive hypercapnia
- •The open-lung approach
- •PEEP
- •Lung recruitment maneuvers
- •Unconventional mechanical ventilation strategies
- •High-frequency percussive ventilation (HFPV)
- •High-frequency oscillatory ventilation
- •Airway pressure release ventilation (APRV)
- •Ventilator associated pneumonia (VAP)
- •(a) Prevention
- •(b) Treatment
- •References
- •Organ responses and organ support
- •Introduction
- •Burn shock and resuscitation
- •Post-burn hypermetabolism
- •Individual organ systems
- •Central nervous system
- •Peripheral nervous system
- •Pulmonary
- •Cardiovascular
- •Renal
- •Gastrointestinal tract
- •Conclusion
- •References
- •Critical care of thermally injured patient
- •Introduction
- •Oxidative stress control strategies
- •Fluid and cardiovascular management beyond 24 hours
- •Other organ function/dysfunction and support
- •The nervous system
- •Respiratory system and inhalation injury
- •Renal failure and renal replacement therapy
- •Gastro-intestinal system
- •Glucose control
- •Endocrine changes
- •Stress response (Fig. 2)
- •Low T3 syndrome
- •Gonadal depression
- •Thermal regulation
- •Metabolic modulation
- •Propranolol
- •Oxandrolone
- •Recombinant human growth hormone
- •Insulin
- •Electrolyte disorders
- •Sodium
- •Chloride
- •Calcium, phosphate and magnesium
- •Calcium
- •Bone demineralization and osteoporosis
- •Micronutrients and antioxidants
- •Thrombosis prophylaxis
- •Conclusion
- •References
- •Treatment of infection in burns
- •Introduction
- •Clinical management strategies
- •Pathophysiology of the burn wound
- •Burn wound infection
- •Cellulitis
- •Impetigo
- •Catheter related infections
- •Urinary tract infection
- •Tracheobronchitis
- •Pneumonia
- •Sepsis in the burn patient
- •The microbiology of burn wound infection
- •Sources of organisms
- •Gram-positive organisms
- •Gram-negative organisms
- •Infection control
- •Pharmacological considerations in the treatment of burn infections
- •Topical antimicrobial treatment
- •Systemic antimicrobial treatment (Table 3)
- •Gram-positive bacterial infections
- •Enterococcal bacterial infections
- •Gram-negative bacterial infections
- •Treatment of yeast and fungal infections
- •The Polyenes (Amphotericin B)
- •Azole antifungals
- •Echinocandin antifungals
- •Nucleoside analog antifungal (Flucytosine)
- •Conclusion
- •References
- •Acute treatment of severely burned pediatric patients
- •Introduction
- •Initial management of the burned child
- •Fluid resuscitation
- •Sepsis
- •Inhalation injury
- •Burn wound excision
- •Burn wound coverage
- •Metabolic response and nutritional support
- •Modulation of the hormonal and endocrine response
- •Recombinant human growth hormone
- •Insulin-like growth factor
- •Oxandrolone
- •Propranolol
- •Glucose control
- •Insulin
- •Metformin
- •Novel therapeutic options
- •Long-term responses
- •Conclusion
- •References
- •Adult burn management
- •Introduction
- •Epidemiology and aetiology
- •Pathophysiology
- •Assessment of the burn wound
- •Depth of burn
- •Size of the burn
- •Initial management of the burn wound
- •First aid
- •Burn blisters
- •Escharotomy
- •General care of the adult burn patient
- •Biological/Semi biological dressings
- •Topical antimicrobials
- •Biological dressings
- •Other dressings
- •Exposure
- •Deep partial thickness wound
- •Total wound excision
- •Serial wound excision and conservative management
- •Full thickness burns
- •Excision and autografting
- •Topical antimicrobials
- •Large full thickness burns
- •Serial excision
- •Mixed depth burn
- •Donor sites
- •Techniques of wound excision
- •Blood loss
- •Antibiotics
- •Anatomical considerations
- •Skin replacement
- •Autograft
- •Allograft
- •Other skin replacements
- •Cultured skin substitutes
- •Skin graft take
- •Rehabilitation and outcome
- •Future care
- •References
- •Burns in older adults
- •Introduction
- •Burn injury epidemiology
- •Pathophysiologic changes and implications for burn therapy
- •Aging
- •Comorbidities
- •Acute management challenges
- •Fluid resuscitation
- •Burn excision
- •Pain and sedation
- •End of life decisions
- •Summary of key points and recommendations
- •References
- •Acute management of facial burns
- •Introduction
- •Anatomy and pathophysiology
- •Management
- •General approach
- •Airway management
- •Facial burn wound management
- •Initial wound care
- •Topical agents
- •Biological dressings
- •Surgical burn wound excision of the face
- •Wound closure
- •Special areas and adjacent of the face
- •Eyelids
- •Nose and ears
- •Lips
- •Scalp
- •The neck
- •Catastrophic injury
- •Post healing rehabilitation and scar management
- •Outcome and reconstruction
- •Summary
- •References
- •Hand burns
- •Introduction
- •Initial evaluation and history
- •Initial wound management
- •Escharotomy and fasciotomy
- •Surgical management: Early excision and grafting
- •Skin substitutes
- •Amputation
- •Hand therapy
- •Secondary reconstruction
- •References
- •Treatment of burns – established and novel technology
- •Introduction
- •Partial thickness burns
- •Biological membranes – amnion and others
- •Xenograft
- •Full thickness burns
- •Dermal analogs
- •Keratinocyte coverage
- •Facial transplantation
- •Tissue engineering and stem cells
- •Gene therapy and growth factors
- •Conclusion
- •References
- •Wound healing
- •History of wound care
- •Types of wounds
- •Mechanisms of wound healing
- •Hemostasis
- •Proliferation
- •Epithelialization
- •Remodeling
- •Fetal wound healing
- •Stem cells
- •Abnormal wound healing
- •Impaired wound healing
- •Hypertrophic scars and keloids
- •Chronic non-healing wounds
- •Conclusions
- •References
- •Pain management after burn trauma
- •Introduction
- •Pathophysiology of pain after burn injuries
- •Nociceptive pain
- •Neuropathic pain
- •Sympathetically Maintained Pain (SMP)
- •Pain rating and documentation
- •Pain management and analgesics
- •Pharmacokinetics in severe burns
- •Form of administration [21]
- •Non-opioids (Table 1)
- •Paracetamol
- •Metamizole
- •Non-steroidal antirheumatics (NSAID)
- •Selective cyclooxygenasis-2-inhibitors
- •Opioids (Table 2)
- •Weak opioids
- •Strong opioids
- •Other analgesics
- •Ketamine (see also intensive care unit and analgosedation)
- •Anticonvulsants (Gabapentin and Pregabalin)
- •Antidepressants with analgesic effects
- •Regional anesthesia
- •Pain management without analgesics
- •Adequate communication
- •Psychological techniques [65]
- •Transcutaneous electrical nerve stimulation (TENS)
- •Particularities of burn pain
- •Wound pain
- •Breakthrough pain
- •Intervention-induced pain
- •Necrosectomy and skin grafting
- •Dressing change of large burn wounds and removal of clamps in skin grafts
- •Dressing change in smaller burn wounds, baths and physical therapy
- •Postoperative pain
- •Mental aspects
- •Intensive care unit
- •Opioid-induced hyperalgesia and opioid tolerance
- •Hypermetabolism
- •Psychic stress factors
- •Risk of infection
- •Monitoring [92]
- •Sedation monitoring
- •Analgesia monitoring (see Fig. 2)
- •Analgosedation (Table 3)
- •Sedation
- •Analgesia
- •References
- •Nutrition support for the burn patient
- •Background
- •Case presentation
- •Patient selection: Timing and route of nutritional support
- •Determining nutritional demands
- •What is an appropriate initial nutrition plan for this patient?
- •Formulations for nutritional support
- •Monitoring nutrition support
- •Optimal monitoring of nutritional status
- •Problems and complications of nutritional support
- •Conclusion
- •References
- •HBO and burns
- •Historical development
- •Contraindications for the use of HBO
- •Conclusion
- •References
- •Nursing management of the burn-injured person
- •Introduction
- •Incidence
- •Prevention
- •Pathophysiology
- •Severity factors
- •Local damage
- •Fluid and electrolyte shifts
- •Cardiovascular, gastrointestinal and renal system manifestations
- •Types of burn injuries
- •Thermal
- •Chemical
- •Electrical
- •Smoke and inhalation injury
- •Clinical manifestations
- •Subjective symptoms
- •Possible complications
- •Clinical management
- •Non-surgical care
- •Surgical care
- •Coordination of care: Burn nursing’s unique role
- •Nursing interventions: Emergent phase
- •Nursing interventions: Acute phase
- •Nursing interventions: Rehabilitative phase
- •Ongoing care
- •Infection prevention and control
- •Rehabilitation medicine
- •Nutrition
- •Pharmacology
- •Conclusion
- •References
- •Outpatient burn care
- •Introduction
- •Epidemiology
- •Accident causes
- •Care structures
- •Indications for inpatient treatment
- •Patient age
- •Total burned body surface area (TBSA)
- •Depth of the burn
- •Pre-existing conditions
- •Accompanying injuries
- •Special injuries
- •Treatment
- •Initial treatment
- •Pain therapy
- •Local treatment
- •Course of treatment
- •Complications
- •Infections
- •Follow-up care
- •References
- •Non-thermal burns
- •Electrical injury
- •Introduction
- •Pathophysiology
- •Initial assessment and acute care
- •Wound care
- •Diagnosis
- •Low voltage injuries
- •Lightning injuries
- •Complications
- •References
- •Symptoms, diagnosis and treatment of chemical burns
- •Chemical burns
- •Decontamination
- •Affection of different organ systems
- •Respiratory tract
- •Gastrointestinal tract
- •Hematological signs
- •Nephrologic symptoms
- •Skin
- •Nitric acid
- •Sulfuric acid
- •Caustic soda
- •Phenol
- •Summary
- •References
- •Necrotizing and exfoliative diseases of the skin
- •Introduction
- •Necrotizing diseases of the skin
- •Cellulitis
- •Staphylococcal scalded skin syndrome
- •Autoimmune blistering diseases
- •Epidermolysis bullosa acquisita
- •Necrotizing fasciitis
- •Purpura fulminans
- •Exfoliative diseases of the skin
- •Stevens-Johnson syndrome
- •Toxic epidermal necrolysis
- •Conclusion
- •References
- •Frostbite
- •Mechanism
- •Risk factors
- •Causes
- •Diagnosis
- •Treatment
- •Rewarming
- •Surgery
- •Sympathectomy
- •Vasodilators
- •Escharotomy and fasciotomy
- •Prognosis
- •Research
- •References
- •Subject index
Outpatient burn care
Bernd Hartmann1, Christian Ottomann2
1Zentrum für Schwerbrandverletzte mit Plastischer Chirurgie, Unfallkrankenhaus Berlin, Germany
2Plastische Chirurgie und Handchirurgie, Intensiveinheit für Schwerbrandverletzte, Universitätsklinikum Schleswig Holstein Campus Lübeck, Germany
Introduction
Epidemiology
Burns are one of the most frequently-occurring types of injuries. Estimates place the number of burn victims in the United States at 1.25 million annually, 40,000 of which require inpatient treatment at a hospital [1]. Other sources write that the US has 700,000 burn victims annually, with 35,000 hospital visits. Both sources show that the percentage of victims with severe injuries requiring inpatient care is around 5%. The figures are similar in Germany. With a total of 350,000 burn victims and 15,000 inpatient cases, the ratio here is 4.3% [2].
Accident causes
The most frequent cause of all injuries from scald burns and burns resulting from contact with flames and hot objects are accidents around the home. The number of such injuries which occur in the workplace has dropped significantly. In this context, most injuries occur in the food service industry as a result of scald burns and contact burns [3].
Care structures
The care structures in highly-developed industrialized nations are, to a large extent, the same. Patients
Marc G. Jeschke et al. (eds.), Handbook of Burns
are offered both outpatient as well as inpatient treatment. In Germany, hospitals’ emergency departments and doctors in private practice are the first point of contact for burn victims. In addition to correctly assessing and evaluating the wound, this is where the patients’ further course of treatment is determined.
The goal of the following chapters is to explain the indications for inpatient burn treatment as well identify the most important care principles. If, in the course of providing inpatient care, complications such as an infection or delayed wound healing should arise, the approach to inpatient treatment must but reconsidered and the patient must be submitted to a specialized burn care facility. In the cooperation between specialized outpatient and inpatient care structures, this is the only way to achieve ideal treatment results for the patient while simultaneously cutting healthcare costs [4].
Indications for inpatient treatment
Upon receiving a patient with a burn injury, the physician first treating the patient at a hospital’s emergency department or in their own practice must determine whether an indication for inpatient treatment exists. In doing so, the physician must initially determine whether the indication for transferring the patient to a specialized center for severe burn
431
© Springer-Verlag/Wien 2012
B. Hartmann, Ch. Ottomann
treatment exists as set forth in the guidelines published by the German Association of Burn Medicine (Deutsche Gesellschaft für Verbrennungsmedizin). These indications (Table 1) must be adhered to in order to begin providing the patient with burn treatment in a timely and professional manner. In addition to the indications set forth in these guidelines which govern whether a patient must be transferred to a specialized burn care center, other indications exist which determine whether a patient should be provided inpatient treatment and forgo outpatient care [5]. The following chapters discuss in detail the most important criteria for making this determination in a professional manner. These include the patient’s age, the depth of the burn, the surface area of the body which has been burned, as well as special burn cases. In this context, correctly evaluating the depth of the burn plays a crucial role. In particular, superficial second degree burns represent an area of conservative treatment. In special cases, they are suitable for outpatient care [6].
In this context, physicians must pursue the following goals:
An optimal functional and aesthetic result for the patient when receiving treatment outside of specialized centers
Palliation of physical and mental discomfort
Adequate follow-up care
Changing the treatment regimen in a timely fashion should complications arise
Patient age
The elderly above the age of 70 as well as small children are particularly at risk for injuries from burns. The group of patients between 5 and 20 years of age has the best prospects for recovery, as this group’s 50% mortality rate now applies to cases where 94.5% of the body’s surface area is burned [7]. In contrast, patients over 70 have a 50% mortality rate in cases where burns cover 29.5% of their body [8]. This agerelated risk is also reflected in the aforementioned guidelines governing indications for transferring patients to specialized burn centers. Patients under the age of 8 as well as those over the age of 60 require special care, which includes hospitalization in spe-
Table 1. Partial and full thickness burn ointments
Dressing agent |
Active substance |
Presentation |
Main use |
Advantages |
Disadvantages |
Bacitracin |
Bacitracin |
Ointment |
Superficial burns, |
Gram (+) coverage |
No G(–) or fungal |
|
|
|
skin grafts |
|
coverage |
Polymyxin |
Polymyxin B |
Ointment |
Superficial burns, |
Gram (–) coverage |
No G(+) or fungal |
|
|
|
skin grafts |
|
coverage |
Mycostatin |
Nystatin |
Ointment |
Superficial burns, |
Good fungal coverage |
No bacterial |
|
|
|
skin grafts |
|
coverage |
Silvadene |
Silver sulfadia- |
Ointment |
Deep burns |
Good bacterial and |
Poor eschar |
|
zine |
|
|
fungal coverage, |
penetration, sulfa |
|
|
|
|
painless |
moiety, leucopenia, |
|
|
|
|
|
pseudoeschar |
|
|
|
|
|
formation |
Sulfamylon |
Mafenide acetate |
Ointment and |
Deep burns |
Good bacterial |
Painful, poor |
|
|
liquid solution |
|
coverage, good eschar |
fungal coverage, |
|
|
|
|
penetration |
metabolic acidosis |
Dakin’s |
Sodium |
Liquid solution |
Superficial and |
Good bacterial |
Very short half life |
|
hypochlorite |
|
deep burns |
coverage, inexpensive |
|
|
|
|
|
and readily available |
|
Silver |
Silver nitrate, |
Liquid solution, |
Superficial burns |
Good bacterial |
Hyponatremia, |
|
silver ion |
dressing sheets |
|
coverage, painless |
dark staining of |
|
|
|
|
|
wounds and linens |
432
Outpatient burn care
cialized centers. Patients above the age of 60 particularly exhibit a slowed biological wound healing process. This must be taken into consideration when making the necessary plans for outpatient treatment. Similarly, small children and the elderly also often have thinner skin [9].
Age represents an important factor for deciding whether a person requires conservative outpatient treatment or inpatient care.
Total burned body surface area (TBSA)
Ultimately, the extent of the surface of the body affected is the deciding factor which determines whether a person can be offered outpatient treatment. When making this estimation, Wallace’s wellknown “Rule of Nines” as well as the “Rule of Palms”, which is particularly suited for estimating the size of smaller injuries, are both used. This rule states that the surface of the patient’s palm represents 1% of their body surface area. When initially evaluating a patient’s burns, one must always estimate the size of the affected area [10].
Depending on the location of the burns, patients with superficial burns covering up to 10% of the body can be given outpatient treatment. The location of the burn is extremely important, however. A completely burned hand or a 1% burn in the face, covering large joints, in the genital area, or near the feet is enough to cause numerous complications, and such burns can heal badly. In such a case, inpatient treatment for patients with smaller wounds in these areas must be considered, and the patients should be transferred to a specialized center pursuant to the aforementioned guidelines [11].
Depth of the burn
Under conservative treatment, individual superficial burns heal with a positive aesthetic and functional result within a maximum period of three weeks. The different burn depths from first to third degree have already been explained in one of this book’s earlier chapters. It is important to note that when dealing with second-degree (partial-thickness) burns, one must differentiate between superficial and deep par- tial-thickness burns. Similar to third-degree burns, deep partial-thickness burns usually also require
Fig. 1. Shows a significantly reddened superficial partialthickness burn
surgery, which means that outpatient treatment is not suitable for patients with such burns [12].
In order to differentiate between the two types of wounds outside of a specialized environment, the following rule can be applied:
Second-degree burns are moist, painful, associated with the formation of blisters and have a coloration from white to pink or red, while thirddegree burns are dry, painless, and have a coloration running from grey-white to brown. Superficial partial-thickness burns exhibit significant capillary regrowth, which means that the skin’s dermal plexus has survived and is enlarged similar to an inflammation. This capillary regrowth is one of the most important signs used to differentiate between superficial and deep partial-thickness burns. At this point however, we must once again refer to the special locations which can also complicate superficial burns, and as such to these zones (see indications for transfer to a burn center). Physicians should select inpatient treatment for those affected by these cases [13,14].
Pre-existing conditions
The burn victim’s pre-existing health condition has both an influence on the healing process as well as on the results which can be expected. Burn patients in reduced states of consciousness, with neurological diseases with paresthesia, as well as those with mental health illnesses should receive inpatient
433
B. Hartmann, Ch. Ottomann
Fig. 2 Shows a deep partial-thickness burn, which from its outward appearance looks similar to a third-degree burn
treatment. The same applies to those taking sedative medications and/or drugs and alcohol [15].
When it comes to internal diseases the focus is on renal insufficiency as well as cardiovascular disorders such as cardiac defects, arrhythmia, high blood pressure and also pulmonary diseases. In addition, this also includes diseases that have an effect on wound healing, such as diabetes mellitus, chronic alcoholism and chronic steroid use. Furthermore, this also includes autoimmune disorders and malignant tumors [16].
When determining a patient’s medical history, these individual diseases must be analyzed and their impact on the entire burned area as well as the patient’s overall condition must be estimated. A person can only receive inpatient treatment after a reliable estimation and risk analysis has been carried out. Frequently analyzing such a patient’s overall health condition may also be necessary during the course of providing inpatient treatment.
Accompanying injuries
The most frequent injury accompanying burns is damage to the pulmonary system as a result of smoke and carbon monoxide. This is known as inhalation trauma and can always occur alongside burns from flames. In this case, the physician providing initial treatment must determine whether the patient has suffered a smoke inhalation injury, and if so, the ex-
tent of the damage [17]. An examination of the patient using auscultation as well as a blood gas analysis are obligatory. Carbon monoxide poisoning can particularly cause the patient to appear disoriented, tired, or exhibit signs of other psychological or mental impairments. Determining the level of carbon monoxide in the patient’s blood is also necessary. It is not enough to simply measure the oxygenation of the patient’s hemoglobin.
If there is reason to suspect that the patient’s upper respiratory tract has been injured through heat or a relevant smoke inhalation injury, they must receive inpatient treatment, and if they only exhibit signs of inhalation trauma, they should at least be kept in the hospital for observation [18].
An additional point of interest are accident-re- lated accompanying injuries. The physician providing initial treatment must always search for additional injuries that may have been caused by falling, jumping from significant heights or trauma caused by the force of explosions. If findings are inconclusive or the mechanics of the accident require it, this case also requires clarification by a team specialized in dealing with such trauma [19].
Special injuries
Finally, one must respond to special types of injuries which are also known to lead to complications during the wound healing process, and as a result, require the injured person to receive inpatient care. This includes electrical burns, chemical burns, as well as self-inflicted injuries or those caused by third parties [20].
Electricity
In day-to-day life, we usually deal with high-voltage electricity of over 1,000 volts or regular household electricity which has between 110 and 220 volts. While injuries from high-voltage electricity or electric arcs can cause significant tissue damage, contact with household electricity often causes heart conditions from arrhythmia to ventricular fibrillation [21].
Since the electrical resistance of the skin is relatively high, the electrical current often looks for other paths through the body. It flows under the skin through neurovascular bundles as well as muscles
434