- •Preface
- •List of contributers
- •History, epidemiology, prevention and education
- •A history of burn care
- •“Black sheep in surgical wards”
- •Toxaemia, plasmarrhea, or infection?
- •The Guinea Pig Club
- •Burns and sulfa drugs at Pearl Harbor
- •Burn center concept
- •Shock and resuscitation
- •Wound care and infection
- •Burn surgery
- •Inhalation injury and pulmonary care
- •Nutrition and the “Universal Trauma Model”
- •Rehabilitation
- •Conclusions
- •References
- •Epidemiology and prevention of burns throughout the world
- •Introduction
- •Epidemiology
- •The inequitable distribution of burns
- •Cost by age
- •Cost by mechanism
- •Limitations of data
- •Risk factors
- •Socioeconomic factors
- •Race and ethnicity
- •Age-related factors: children
- •Age-related factors: the elderly
- •Regional factors
- •Gender-related factors
- •Intent
- •Comorbidity
- •Agents
- •Non-electric domestic appliances
- •War, mass casualties, and terrorism
- •Interventions
- •Smoke detectors
- •Residential sprinklers
- •Hot water temperature regulation
- •Lamps and stoves
- •Fireworks legislation
- •Fire-safe cigarettes
- •Children’s sleepwear
- •Acid assaults
- •Burn care systems
- •Role of the World Health Organization
- •Conclusions and recommendations
- •Surveillance
- •Smoke alarms
- •Gender inequality
- •Community surveys
- •Acknowledgements
- •References
- •Prevention of burn injuries
- •Introduction
- •Burns prevalence and relevance
- •Burn injury risk factors
- •WHERE?
- •Burn prevention types
- •Burn prevention: The basics to design a plan
- •Flame burns
- •Prevention of scald burns
- •Conclusions
- •References
- •Burns associated with wars and disasters
- •Introduction
- •Wartime burns
- •Epidemiology of burns sustained during combat operations
- •Fluid resuscitation and initial burn care in theater
- •Evacuation of thermally-injured combat casualties
- •Care of host-nation burn patients
- •Disaster-related burns
- •Epidemiology
- •Treatment of disaster-related burns
- •The American Burn Association (ABA) disaster management plan
- •Summary
- •References
- •Education in burns
- •Introduction
- •Surgical education
- •Background
- •Simulation
- •Education in the internet era
- •Rotations as courses
- •Mentorship
- •Peer mentorship
- •Hierarchical mentorship
- •What is a mentor
- •Implementation
- •Interprofessional education
- •What is interprofessional education
- •Approaches to interprofessional education
- •References
- •European practice guidelines for burn care: Minimum level of burn care provision in Europe
- •Foreword
- •Background
- •Introduction
- •Burn injury and burn care in general
- •Conclusion
- •References
- •Pre-hospital and initial management of burns
- •Introduction
- •Modern care
- •Early management
- •At the accident
- •At a local hospital – stabilization prior to transport to the Burn Center
- •Transportation
- •References
- •Medical documentation of burn injuries
- •Introduction
- •Medical documentation of burn injuries
- •Contents of an up-to-date burns registry
- •Shortcomings in existing documentation systems designs
- •Burn depth
- •Burn depth as a dynamic process
- •Non-clinical methods to classify burn depth
- •Burn extent
- •Basic principles of determining the burn extent
- •Methods to determine burn extent
- •Computer aided three-dimensional documentation systems
- •Methods used by BurnCase 3D
- •Creating a comparable international database
- •Results
- •Conclusion
- •Financing and accomplishment
- •References
- •Pathophysiology of burn injury
- •Introduction
- •Local changes
- •Burn depth
- •Burn size
- •Systemic changes
- •Hypovolemia and rapid edema formation
- •Altered cellular membranes and cellular edema
- •Mediators of burn injury
- •Hemodynamic consequences of acute burns
- •Hypermetabolic response to burn injury
- •Glucose metabolism
- •Myocardial dysfunction
- •Effects on the renal system
- •Effects on the gastrointestinal system
- •Effects on the immune system
- •Summary and conclusion
- •References
- •Anesthesia for patients with acute burn injuries
- •Introduction
- •Preoperative evaluation
- •Monitors
- •Pharmacology
- •Postoperative care
- •References
- •Diagnosis and management of inhalation injury
- •Introduction
- •Effects of inhaled gases
- •Carbon monoxide
- •Cyanide toxicity
- •Upper airway injury
- •Lower airway injury
- •Diagnosis
- •Resuscitation after inhalation injury
- •Other treatment issues
- •Prognosis
- •Conclusions
- •References
- •Respiratory management
- •Airway management
- •(a) Endotracheal intubation
- •(b) Elective tracheostomy
- •Chest escharotomy
- •Conventional mechanical ventilation
- •Introduction
- •Pathophysiological principles
- •Low tidal volume and limited plateau pressure approaches
- •Permissive hypercapnia
- •The open-lung approach
- •PEEP
- •Lung recruitment maneuvers
- •Unconventional mechanical ventilation strategies
- •High-frequency percussive ventilation (HFPV)
- •High-frequency oscillatory ventilation
- •Airway pressure release ventilation (APRV)
- •Ventilator associated pneumonia (VAP)
- •(a) Prevention
- •(b) Treatment
- •References
- •Organ responses and organ support
- •Introduction
- •Burn shock and resuscitation
- •Post-burn hypermetabolism
- •Individual organ systems
- •Central nervous system
- •Peripheral nervous system
- •Pulmonary
- •Cardiovascular
- •Renal
- •Gastrointestinal tract
- •Conclusion
- •References
- •Critical care of thermally injured patient
- •Introduction
- •Oxidative stress control strategies
- •Fluid and cardiovascular management beyond 24 hours
- •Other organ function/dysfunction and support
- •The nervous system
- •Respiratory system and inhalation injury
- •Renal failure and renal replacement therapy
- •Gastro-intestinal system
- •Glucose control
- •Endocrine changes
- •Stress response (Fig. 2)
- •Low T3 syndrome
- •Gonadal depression
- •Thermal regulation
- •Metabolic modulation
- •Propranolol
- •Oxandrolone
- •Recombinant human growth hormone
- •Insulin
- •Electrolyte disorders
- •Sodium
- •Chloride
- •Calcium, phosphate and magnesium
- •Calcium
- •Bone demineralization and osteoporosis
- •Micronutrients and antioxidants
- •Thrombosis prophylaxis
- •Conclusion
- •References
- •Treatment of infection in burns
- •Introduction
- •Clinical management strategies
- •Pathophysiology of the burn wound
- •Burn wound infection
- •Cellulitis
- •Impetigo
- •Catheter related infections
- •Urinary tract infection
- •Tracheobronchitis
- •Pneumonia
- •Sepsis in the burn patient
- •The microbiology of burn wound infection
- •Sources of organisms
- •Gram-positive organisms
- •Gram-negative organisms
- •Infection control
- •Pharmacological considerations in the treatment of burn infections
- •Topical antimicrobial treatment
- •Systemic antimicrobial treatment (Table 3)
- •Gram-positive bacterial infections
- •Enterococcal bacterial infections
- •Gram-negative bacterial infections
- •Treatment of yeast and fungal infections
- •The Polyenes (Amphotericin B)
- •Azole antifungals
- •Echinocandin antifungals
- •Nucleoside analog antifungal (Flucytosine)
- •Conclusion
- •References
- •Acute treatment of severely burned pediatric patients
- •Introduction
- •Initial management of the burned child
- •Fluid resuscitation
- •Sepsis
- •Inhalation injury
- •Burn wound excision
- •Burn wound coverage
- •Metabolic response and nutritional support
- •Modulation of the hormonal and endocrine response
- •Recombinant human growth hormone
- •Insulin-like growth factor
- •Oxandrolone
- •Propranolol
- •Glucose control
- •Insulin
- •Metformin
- •Novel therapeutic options
- •Long-term responses
- •Conclusion
- •References
- •Adult burn management
- •Introduction
- •Epidemiology and aetiology
- •Pathophysiology
- •Assessment of the burn wound
- •Depth of burn
- •Size of the burn
- •Initial management of the burn wound
- •First aid
- •Burn blisters
- •Escharotomy
- •General care of the adult burn patient
- •Biological/Semi biological dressings
- •Topical antimicrobials
- •Biological dressings
- •Other dressings
- •Exposure
- •Deep partial thickness wound
- •Total wound excision
- •Serial wound excision and conservative management
- •Full thickness burns
- •Excision and autografting
- •Topical antimicrobials
- •Large full thickness burns
- •Serial excision
- •Mixed depth burn
- •Donor sites
- •Techniques of wound excision
- •Blood loss
- •Antibiotics
- •Anatomical considerations
- •Skin replacement
- •Autograft
- •Allograft
- •Other skin replacements
- •Cultured skin substitutes
- •Skin graft take
- •Rehabilitation and outcome
- •Future care
- •References
- •Burns in older adults
- •Introduction
- •Burn injury epidemiology
- •Pathophysiologic changes and implications for burn therapy
- •Aging
- •Comorbidities
- •Acute management challenges
- •Fluid resuscitation
- •Burn excision
- •Pain and sedation
- •End of life decisions
- •Summary of key points and recommendations
- •References
- •Acute management of facial burns
- •Introduction
- •Anatomy and pathophysiology
- •Management
- •General approach
- •Airway management
- •Facial burn wound management
- •Initial wound care
- •Topical agents
- •Biological dressings
- •Surgical burn wound excision of the face
- •Wound closure
- •Special areas and adjacent of the face
- •Eyelids
- •Nose and ears
- •Lips
- •Scalp
- •The neck
- •Catastrophic injury
- •Post healing rehabilitation and scar management
- •Outcome and reconstruction
- •Summary
- •References
- •Hand burns
- •Introduction
- •Initial evaluation and history
- •Initial wound management
- •Escharotomy and fasciotomy
- •Surgical management: Early excision and grafting
- •Skin substitutes
- •Amputation
- •Hand therapy
- •Secondary reconstruction
- •References
- •Treatment of burns – established and novel technology
- •Introduction
- •Partial thickness burns
- •Biological membranes – amnion and others
- •Xenograft
- •Full thickness burns
- •Dermal analogs
- •Keratinocyte coverage
- •Facial transplantation
- •Tissue engineering and stem cells
- •Gene therapy and growth factors
- •Conclusion
- •References
- •Wound healing
- •History of wound care
- •Types of wounds
- •Mechanisms of wound healing
- •Hemostasis
- •Proliferation
- •Epithelialization
- •Remodeling
- •Fetal wound healing
- •Stem cells
- •Abnormal wound healing
- •Impaired wound healing
- •Hypertrophic scars and keloids
- •Chronic non-healing wounds
- •Conclusions
- •References
- •Pain management after burn trauma
- •Introduction
- •Pathophysiology of pain after burn injuries
- •Nociceptive pain
- •Neuropathic pain
- •Sympathetically Maintained Pain (SMP)
- •Pain rating and documentation
- •Pain management and analgesics
- •Pharmacokinetics in severe burns
- •Form of administration [21]
- •Non-opioids (Table 1)
- •Paracetamol
- •Metamizole
- •Non-steroidal antirheumatics (NSAID)
- •Selective cyclooxygenasis-2-inhibitors
- •Opioids (Table 2)
- •Weak opioids
- •Strong opioids
- •Other analgesics
- •Ketamine (see also intensive care unit and analgosedation)
- •Anticonvulsants (Gabapentin and Pregabalin)
- •Antidepressants with analgesic effects
- •Regional anesthesia
- •Pain management without analgesics
- •Adequate communication
- •Psychological techniques [65]
- •Transcutaneous electrical nerve stimulation (TENS)
- •Particularities of burn pain
- •Wound pain
- •Breakthrough pain
- •Intervention-induced pain
- •Necrosectomy and skin grafting
- •Dressing change of large burn wounds and removal of clamps in skin grafts
- •Dressing change in smaller burn wounds, baths and physical therapy
- •Postoperative pain
- •Mental aspects
- •Intensive care unit
- •Opioid-induced hyperalgesia and opioid tolerance
- •Hypermetabolism
- •Psychic stress factors
- •Risk of infection
- •Monitoring [92]
- •Sedation monitoring
- •Analgesia monitoring (see Fig. 2)
- •Analgosedation (Table 3)
- •Sedation
- •Analgesia
- •References
- •Nutrition support for the burn patient
- •Background
- •Case presentation
- •Patient selection: Timing and route of nutritional support
- •Determining nutritional demands
- •What is an appropriate initial nutrition plan for this patient?
- •Formulations for nutritional support
- •Monitoring nutrition support
- •Optimal monitoring of nutritional status
- •Problems and complications of nutritional support
- •Conclusion
- •References
- •HBO and burns
- •Historical development
- •Contraindications for the use of HBO
- •Conclusion
- •References
- •Nursing management of the burn-injured person
- •Introduction
- •Incidence
- •Prevention
- •Pathophysiology
- •Severity factors
- •Local damage
- •Fluid and electrolyte shifts
- •Cardiovascular, gastrointestinal and renal system manifestations
- •Types of burn injuries
- •Thermal
- •Chemical
- •Electrical
- •Smoke and inhalation injury
- •Clinical manifestations
- •Subjective symptoms
- •Possible complications
- •Clinical management
- •Non-surgical care
- •Surgical care
- •Coordination of care: Burn nursing’s unique role
- •Nursing interventions: Emergent phase
- •Nursing interventions: Acute phase
- •Nursing interventions: Rehabilitative phase
- •Ongoing care
- •Infection prevention and control
- •Rehabilitation medicine
- •Nutrition
- •Pharmacology
- •Conclusion
- •References
- •Outpatient burn care
- •Introduction
- •Epidemiology
- •Accident causes
- •Care structures
- •Indications for inpatient treatment
- •Patient age
- •Total burned body surface area (TBSA)
- •Depth of the burn
- •Pre-existing conditions
- •Accompanying injuries
- •Special injuries
- •Treatment
- •Initial treatment
- •Pain therapy
- •Local treatment
- •Course of treatment
- •Complications
- •Infections
- •Follow-up care
- •References
- •Non-thermal burns
- •Electrical injury
- •Introduction
- •Pathophysiology
- •Initial assessment and acute care
- •Wound care
- •Diagnosis
- •Low voltage injuries
- •Lightning injuries
- •Complications
- •References
- •Symptoms, diagnosis and treatment of chemical burns
- •Chemical burns
- •Decontamination
- •Affection of different organ systems
- •Respiratory tract
- •Gastrointestinal tract
- •Hematological signs
- •Nephrologic symptoms
- •Skin
- •Nitric acid
- •Sulfuric acid
- •Caustic soda
- •Phenol
- •Summary
- •References
- •Necrotizing and exfoliative diseases of the skin
- •Introduction
- •Necrotizing diseases of the skin
- •Cellulitis
- •Staphylococcal scalded skin syndrome
- •Autoimmune blistering diseases
- •Epidermolysis bullosa acquisita
- •Necrotizing fasciitis
- •Purpura fulminans
- •Exfoliative diseases of the skin
- •Stevens-Johnson syndrome
- •Toxic epidermal necrolysis
- •Conclusion
- •References
- •Frostbite
- •Mechanism
- •Risk factors
- •Causes
- •Diagnosis
- •Treatment
- •Rewarming
- •Surgery
- •Sympathectomy
- •Vasodilators
- •Escharotomy and fasciotomy
- •Prognosis
- •Research
- •References
- •Subject index
A. Arno, J. Knighton
Table 1. Estimated number of deaths and mortality rates due to fire-related burn by WHO region and Income group, 2002
REGION |
Africa |
America |
South- |
Europe |
Eastern |
Western Pacific |
World |
||||
|
|
|
|
East |
|
|
Mediterranean |
|
|
|
|
|
|
|
|
Asia |
|
|
|
|
|
|
|
Income group |
L/M |
H |
L/M |
L/M |
H |
L/M |
H |
L/M |
H |
L/M |
|
Number of burn deaths |
4.3 |
4 |
4 |
184 |
3 |
21 |
0,1 |
32 |
2 |
18 |
312 |
(thousands) |
|
|
|
|
|
|
|
|
|
|
|
Death rate (per 100 000 |
6.2 |
1.2 |
0.8 |
11.6 |
0.7 |
4.5 |
0.9 |
6.4 |
1.2 |
1.2 |
5 |
population) |
|
|
|
|
|
|
|
|
|
|
|
Proportion global |
13.8 |
1.3 |
1.3 |
59 |
1 |
6.7 |
0.02 |
10.3 |
0.6 |
5.8 |
100 |
mortality due to fires (%) |
|
|
|
|
|
|
|
|
|
|
|
L = Low, M = Middle, H = High.
From: WHO Global Burden of Disease Database, 2002 (Version 5)
affecting nearly every organ system and leading to significant morbidity and mortality. In order to maintain a morbidity and mortality statistics database for burns, the ABA/TRACS (Trauma Registry of the American College of Surgeons) Burn Patient Registry was created in the US in the 1990s. However, there is still a strong need for homogeneous national and international burn registration systems.
Although mortality has declined over the past few years due to improved medical care and promotion of burn prevention, management of major burns still remains a challenge, even in modern, developed countries. The increasing number of burn survivors are at risk for developing long-term psychological and physical sequelae, with potentially devastating consequences to them, their families and society in general. Furthermore, burns have important human and material costs. Providing adequate hospital care to a burn patient costs approximately US$ 1000 per day and major burns usually require admission to intensive care units, which are very resource-inten- sive expensive.
In most developing countries, the same standard of care is not possible due to limited resources and inaccessibility to sophisticated skills and technologies, leading to a higher morbidity and mortality. Although these countries are in special need of burn prevention, the reality is often the opposite, where there is a lack of government-funded burn prevention programmes. In these countries, some health authorities consider injury prevention to have a much lower priority than disease prevention. Despite mounting evidence that injury is largely pre-
ventable, burn prevention has been forgotten in the past by public health authorities as these injuries are believed to be random accidents. While natural disasters are unavoidable, accidents can be prevented and represent a public health problem that requires government and community involvement. A key to keep in mind is that the best care for burns is prevention, which takes time, energy and money. Eventually, it is likely the most effective solution to the world’s burn problem.
Burn injury risk factors
Burn risk is linked with poverty, lack of running water, crowding, illiteracy, unemployment, lapses in child supervision – mostly in large and single-parent families, recent pregnancies or mothers’ being away from home, prior history of a sibling being burned-, presence of pre-existing physical/emotional challenges in a child and lack of education (although in some societies, higher education of individuals is associated with increased risk of immolation rituals involving burns, for example).
With these risk factors in mind, one can ascertain why burns are more common in the developing world and how low socioeconomic status has been linked to increased risk of unintentional injury and mortality.
Another important risk factor for burns is drug use. Medications, such as sleeping pills, narcotics, synthetic stimulant drugs, such as methamphetamines, alcohol and/or smoking are usually the pro-
62
Prevention of burn injuries
drome of flame burns in many teenagers and adults. For instance, in South Korea it is not unusual to see groups of teenagers in a room engaging in butane gas abuse, which causes euphoria and hallucinations. Facial burns result from sniffing the flammable gas, in addition to potentially severe systemic effects, such as pulmonary oedema, gastritis and cardiac dysrhythmias. Sniffing nitrous oxide also causes confusion; in this case, the cold gas results in frostbite to the cheek.
In a large European review, risk factors for death from a burn were found to be older age, higher total body surface area burned and previous comorbidity in the form of chronic diseases. Multiorgan failure and sepsis were the most frequent leading causes of death. Burn shock and inhalation injury were responsible for most of the early deaths (< 48h). Half of the burns were suffered by children < 16 years old and 60% occurred in men (but in elderly people, women were more frequently burned).
When dealing with burn risk factors, it is important to analyze the who, how and where factors in order to design an effective prevention plan.
Table 2. Elderly burn prevention tips
WHO?
Regarding the age of the individual, children under 5 years of age comprise the highest risk group for burn injuries. Causative factors in all incidents involving babies are a mixture of imprudence, impulsiveness, curiosity, lack of experience and a desire to imitate adults. Furthermore, this age group lacks a sense of danger and awareness and they hardly understand cause-and-effect relationships.
At the other end of the age continuum, elderly people over 64 years are also a high risk population. Their risk of dying in a fire is 2.5 times greater than the general population. The main causes of burn injuries in the elderly are flame burns due to smoking and cooking (Table 2). Additional risk factors include medical conditions associated with physical or mental impairment: stroke, poor eyesight, decreased hearing and mobility, diabetes (peripheral neuropathy with decreased or no lower extremity pain perception), dementia (such as Alzheimer’s, with confusion and forgetfulness), depression and suicide.
FLAME burns |
1. Ask a relative or neighbour to routinely check for gas leak odor. |
||
|
2. |
Use large ashtrays. Smoke only while upright. Never smoke in bed or when drowsy. |
|
|
3. |
Never use flammable liquids to start a fire or prime a carburetor or as a cleaning solvent. |
|
|
4. |
Never store flammable liquids near a pilot light or other heat source. |
|
|
5 |
Check the smoke detector battery once a month. Use a broom handle to perform the check or ask a |
|
|
|
friend to do so. |
|
|
6 |
Have a flashlight, keys, eyeglasses and whistle at the bedside to summon help if needed. |
7.Wear close-fitting clothes while cooking or near any potentially dangerous heat source (fireplace, campfire, wood-burning stove). Garments that are flame resistant are recommended.
8.Use the back burners of the stove and turn handles inward.
9.Avoid throw rugs in the kitchen area and keep the floor clean to avoid falls.
10.Use a cooking timer with an audible alarm.
11.If using a speace heater, ensure that the automatic shut off is in working order should the heater accidentally tip over.
12.Never lay anything on or near a heating device (e.g., space heater, wood-burning stove, kitchen stove, baseboard heater).
CONTACT burns |
1. |
Use all heating devices that are placed on or near the skin with caution (e.g., heating pad, hot water |
|
|
bottles, space heaters). |
SCALD burns |
1. |
Place a nonskid mat and handrails in the bathtub or shower to prevent accidental falls and to allow |
|
|
easy access in and out of the area. |
|
2. Check the temperature on the hot water heater; the recommended setting is 120ºF (48.8ºC). Install |
|
|
|
antiscald devices in bathroom plumbing. |
Adapted from: Thompson RM, Carrougher GJ (1998) Burn prevention. In: Carrougher GJ (ed) Burn care and therapy. Mosby, St. Louis, pp 497–524 [18]
63
A. Arno, J. Knighton
Table 3. Burn prevention tips at home: in the kitchen, living room and bathroom
KITCHEN |
1. Keep hot items in the centre of the table and away from children. |
2.Keep food away from the stove, so no one will be tempted to reach across hot stove elements.
3.Keep young children away from the cooking area.
4.Use place mats instead of tablecloths (young children use tablecloths to pull themselves up)
5.Roll up electrical cords and unplug appliances when not in use.
6.Use pot holders, not towels.
7.Turn pot handles, inward, toward the back of the stove; use back elements of stove for cooking.
8.Store pot holders, paper towels and seasonings, candy or toys away from the stove top.
9.Avoid full or puffy sleeves while cooking.
10.Use a large lid or baking soda to put out small grease fires in pans.
LIVING-ROOM |
1. Do not use extension cords in place of permanent wires. |
|
|
2. |
Cover unused electrical outlets with safety plugs. |
|
3. |
Use fireplace matches to light a fireplace. |
|
4. |
Keep matches and lighters away from children. |
|
5. |
Soak cigarettes in water before placing in garbage to ensure they are fully extinguished. |
BATHROOM |
1. Run hot and cold water together. |
|
|
2. |
Set the hot water heater thermostat to low 50ºC. |
|
3. |
Never leave children alone in the bathroom. |
|
4. |
Use a “no slip” plastic mat in the bathtub to prevent falls. |
In India, young women (16–35 years old) are the most prone to suffer burns at home, due to the tradition of cooking at floor level or over an open fire, compounded by the wearing of loose fitting clothing made from non-flame retardant fabric. In the least developed European countries, there is also female burn predominance and a higher proportion of electrical burn injuries comparing with western Europe, with a male predominance.
WHERE?
Most childhood burns occur in the home, in developed as well as in developing countries. The modern home can contain a number of harmful substances and pieces of equipment, such as electricity, gas and chemicals (Table 3). Lapses in child supervision, such as forgetting to turn off the oven or stove element at bath time lead to a higher risk of unintentional burn injuries.
Adult burns are reported to have a more variable location, with almost equal frequencies in the home, outdoors and in the workplace. For all age groups, the kitchen is the most common scene of burns, followed by the bathroom and the outdoors (i. e. backyard, garage).
HOW?
Burns in children are caused usually by scald, flame and contact burns, in order of most to least frequency (although in some countries, such as Australia, contact burns are the second cause after scalds, and, in the developing world, flame burns are the most common). It has been shown that when the child becomes older, flame injury predominates, becoming the most common cause of burns in children aged 6–17 years old.
Not only are young children more prone to suffer contact burns, but also the elderly, the physically impaired people, patients with diabetes, all of whom may have some inability to withdraw from heated objects or who have limited sensation to their extremities (Table 10).
In the developing world, the commonest cause of injury is a flame burn. Most such accidents are related to malfunctioning kerosene pressure stoves and homemade kerosene lamps used for lighting, or from domestic appliances using flammable fuel. On the other hand, many households are made of highly combustible and toxic materials, such as wood and plastics, which burn easily, leading to high mortality rates due to severe smoke inhalation injury.
In the developed world, fire is sometimes related, not only to accidental burns, but also to homi-
64