- •Burn Care and Treatment
- •Contents
- •1.1 Initial Assessment and Emergency Treatment
- •Box 1.1. Primary and Secondary Survey
- •1.2 Fluid Resuscitation and Early Management
- •1.2.1 Fluid Resuscitation
- •1.2.2 Endpoint of Burn Resuscitation
- •1.2.4 Role of Colloids, Hypertonic Saline, and Antioxidants in Resuscitation
- •1.2.4.1 Colloids
- •1.2.4.2 Hypertonic Saline
- •1.2.4.3 Antioxidants: High-Dose Vitamin C
- •1.3 Evaluation and Early Management of Burn Wound
- •1.3.1 Evaluation of Burn Depth
- •1.3.2 Choice of Topical Dressings
- •1.3.3 Escharotomy
- •1.3.4 Operative Management
- •References
- •2: Pathophysiology of Burn Injury
- •2.1 Introduction
- •2.2 Local Changes
- •2.2.1 Temperature and Time Effect
- •2.2.2 Etiology
- •2.2.3 Pathophysiologic Changes
- •2.2.4 Burn Size
- •2.3 Systemic Changes
- •2.3.1 Edema Formation
- •2.3.3.1 Resting Energy Expenditure
- •2.3.3.2 Muscle Catabolism
- •2.3.3.3 Glucose and Lipid Metabolism
- •2.3.4 Renal System
- •2.3.5 Gastrointestinal System
- •2.3.6 Immune System
- •2.4 Summary and Conclusion
- •References
- •3: Wound Healing and Wound Care
- •3.1 Introduction
- •3.2 Physiological Versus Pathophysiologic Wound Healing
- •3.2.1 Transforming Growth Factor Beta
- •3.2.2 Interactions Between Keratinocytes and Fibroblasts
- •3.2.3 Matrix Metalloproteinases (MMP)
- •3.3.1 Burn Wound Excision
- •3.3.2 Burn Wound Coverage
- •3.3.3 Autografts
- •3.3.4 Epidermal Substitutes
- •3.3.5 Dermal Substitutes
- •3.3.6 Epidermal/Dermal Substitutes
- •3.4 Summary
- •References
- •4: Infections in Burns
- •4.1 Burn Wound Infections
- •4.1.1 Diagnosis and Treatment of Burn Wound Infections
- •4.1.1.1 Introduction
- •4.1.2 Common Pathogens and Diagnosis
- •4.1.3 Clinical Management
- •4.1.3.1 Local
- •4.1.3.2 Systemic
- •4.1.4 Conclusion
- •4.4 Guidelines for Sepsis Resuscitation
- •References
- •5: Acute Burn Surgery
- •5.1 Introduction
- •5.2 Burn Wound Evaluation
- •5.3 Escharotomy/Fasciotomy
- •5.4 Surgical Burn Wound Management
- •5.5.1 Face
- •5.5.2 Hands
- •5.6 Treatment Standards in Burns Larger Than Sixty Percent TBSA
- •5.7 Temporary Coverage
- •5.9.1 Early Mobilisation
- •5.9.2 Nutrition and Anabolic Agents
- •Bibliography
- •6.1 Introduction
- •6.2 Initial and Early Hospital Phase
- •6.2.1 Blood Pressure
- •6.2.1.1 Resuscitation
- •6.2.1.2 Albumin
- •6.2.1.3 Transfusion
- •6.2.1.4 Vasopressors
- •6.2.2 Urine Output
- •6.2.4 Respiration
- •6.2.4.1 Ventilation Settings
- •6.2.5 Inhalation Injury
- •6.2.6 Invasive and Noninvasive Thermodilution Catheter (PiCCO Catheter)
- •6.2.7 Serum Organ Markers
- •6.3 Later Hospital Phase
- •6.3.1 Central Nervous System
- •6.3.1.1 Intensive Care Unit-Acquired Weakness
- •6.3.1.2 Thermal Regulation
- •6.3.2 Heart
- •6.3.3 Lung
- •6.3.3.1 Ventilator-Associated Pneumonia
- •6.3.4 Liver/GI
- •6.3.4.1 GI Complications/GI Prophylaxis/Enteral Nutrition
- •6.3.4.2 Micronutrients and Antioxidants
- •6.3.5 Renal
- •6.3.6 Hormonal (Thyroid, Adrenal, Gonadal)
- •6.3.7 Electrolyte Disorders
- •6.3.7.1 Sodium
- •6.3.7.2 Chloride
- •6.3.7.3 Phosphate and Magnesium
- •6.3.7.4 Calcium
- •6.3.8 Bone Demineralization and Osteoporosis
- •6.3.9 Coagulation and Thrombosis Prophylaxis
- •Conclusion
- •References
- •7.1 Introduction
- •7.2.1 Glucose Metabolism
- •7.2.2 Fat Metabolism
- •7.2.3 Protein Metabolism
- •7.3 Attenuation of the Hypermetabolic Response
- •7.3.1.1 Nutrition
- •Nutritional Route
- •Initiation of Nutrition
- •Amount of Nutrition
- •Composition of Nutrition (Table 7.1)
- •7.3.1.2 Early Excision
- •7.3.1.3 Environmental Support
- •7.3.1.4 Exercise and Adjunctive Measures
- •7.3.2 Pharmacologic Modalities
- •7.3.2.1 Recombinant Human Growth Hormone
- •7.3.2.2 Insulin-Like Growth Factor
- •7.3.2.3 Oxandrolone
- •7.3.2.4 Propranolol
- •7.3.2.5 Insulin
- •7.3.2.6 Metformin
- •7.3.2.7 Other Options
- •7.4 Summary and Conclusion
- •References
- •8.1 Introduction
- •8.2 Knowledge Base
- •8.2.1.1 Incidence
- •8.3 Aetiology and Risk Factors
- •8.3.1 Pathophysiology
- •8.3.1.1 Severity Factors
- •Box 8.1. Burn Severity Factors
- •8.3.2 Local Damage
- •8.3.3 Fluid and Electrolyte Shifts
- •8.4 Cardiovascular, Gastrointestinal and Renal System Manifestations
- •8.4.1 Types of Burn Injuries
- •8.4.1.1 Clinical Manifestations
- •Box 8.2. Primary Survey Assessment
- •Box 8.3. Signs and Symptoms of Hypovolemic Shock
- •Box 8.4. Physical Findings of Inhalation Injury
- •Box 8.5. Signs and Symptoms of Vascular Compromise
- •Box 8.6. Secondary Survey Assessment
- •8.5 Clinical Management
- •8.5.1 Nonsurgical Care
- •Box 8.7. Secondary Survey Highlights
- •Box 8.8. First Aid Management at the Scene
- •Box 8.9. Treatment of the Severely Burned Patient on Admission
- •Box 8.10. Fluid Resuscitation Using the Parkland (Baxter) Formula
- •Box 8.11. Properties of Topical Antimicrobial Agents
- •Box 8.12. Criteria for Burn Wound Coverings
- •8.5.2 Surgical Care
- •8.5.3 Pharmacological Support
- •8.5.4 Psychosocial Support
- •References
- •9.1 Electrical Injuries
- •9.1.1 Introduction
- •9.1.2 Diagnosis and Management
- •9.2 Chemical Burns
- •9.3 Cold Injury (Frostbite)
- •References
- •10.1 Introduction
- •10.2 Pathophysiology
- •10.3 Scarring
- •10.4 Therapy
- •10.5 Psychological Aspects
- •10.6 Return to Work
- •10.8 Exercise
- •10.9 Summary
- •References
- •11: Burn Reconstruction Techniques
- •11.1 From the Reconstructive Ladder to the Reconstructive Elevator
- •11.2 The Reconstructive Clockwork
- •11.2.1 General Principles
- •11.3 Indication and Timing of Surgical Intervention
- •11.4 The Techniques of Reconstruction
- •11.4.1 Excision Techniques
- •11.4.1.1 W-Plasty and Geometric Broken Line Closure
- •11.4.2 Serial Excision and Tissue Expansion
- •11.4.3 Skin Grafting Techniques
- •11.4.4 Local Skin Flaps
- •11.4.4.1 Z-Plasty
- •11.4.4.2 Double Opposing Z-Plasty
- •11.4.4.3 ¾ Z-plasty or half-Z
- •11.4.4.4 Musculocutaneous (MC) or Fasciocutaneous (FC) Flap Technique
- •11.4.5 Distant Flaps
- •11.4.5.1 Free Tissue Transfer
- •11.4.5.2 Perforator Flaps
- •11.4.6 Composite Tissue Allotransplantation
- •11.4.7 Regeneration: Tissue Engineering
- •11.4.8 Robotics/Prosthesis
- •11.5 Summary
- •References
- •Appendix
- •Sedatives and Pain Medications
- •Index
112 |
J. Knighton |
|
|
Table 8.1 American Burn Association adult burn classification |
|
Classification |
Assessment criteria |
Minor burn injury |
<15 % TBSA burn in adults <40 years age |
|
<10 % TBSA burn in adults >40 years age |
|
<2 % TBSA full-thickness burn without risk of functional or |
|
aesthetic impairment or disability |
|
|
Moderate uncomplicated |
15–25 % TBSA burn in adults <40 years age |
burn injury |
10–20 % TBSA burn in adults >40 years age |
|
<10 % TBSA full-thickness burn without functional or aesthetic risk |
|
to burns involving the face, eyes, ears, hands, feet or perineum |
Major burn injury |
>25 % TBSA burn in adults <40 years age |
|
>20 % TBSA burn in adults >40 years age |
|
OR >10 % TBSA full-thickness burn (any age) |
|
OR injuries involving the face, eyes, ears, hands, feet |
|
OR perineum likely to result in functional or aesthetic disability |
|
OR high-voltage electrical burn |
|
OR all burns with inhalation injury or major trauma |
8.2.1.2 Classification
Burn complexity can range from a relatively minor, uncomplicated injury to a life-threatening, multisystem trauma. The American Burn Association (ABA) has a useful classification system that rates burn injury magnitude from minor to moderate, uncomplicated to major (Table 8.1).
8.3Aetiology and Risk Factors
The causes of burn injuries are numerous and found in both the home, leisure and workplace settings (Table 8.2).
8.3.1Pathophysiology
8.3.1.1 Severity Factors
There are five factors that need to be considered when determining the severity of a burn injury (Box 8.1):
1.Extent – There are several methods available to accurately calculate the percentage of body surface area involved:
•The simplest is the rule of nines (see Chap. 1, Fig. 1.1 and Chap. 2, Fig. 2.2).
However, it is only for use with the adult burn population.
•The Lund and Browder method (see Chap. 1, Fig. 1.1 and Chap. 2, Fig. 2.2) is useful for all age groups, but is more complicated to use.
•There is a paediatric version of the Lund and Browder method (see Chap. 2, Fig. 2.2).
8 Nursing Management of the Burn-Injured Person |
113 |
|
|
Table 8.2 Causes of burn injuries |
|
Home and leisure |
Workplace |
Hot water heaters set too high (140 °F or 60 °C) |
Electricity: |
Overloaded electrical outlets |
Power lines |
Frayed electrical wiring |
Outlet boxes |
Carelessness with cigarettes, lighters, matches, candles |
Chemicals: |
Pressure cookers |
Acids |
Microwaved foods and liquids |
Alkalis |
Hot grease or cooking liquids |
Tar |
Open space heaters |
Hot steam sources: |
Gas fireplace doors |
Boilers |
Radiators |
Pipes |
Hot sauna rocks |
Industrial cookers |
Improper use of flammable liquids: |
Hot industrial presses |
Starter fluids |
Flammable liquids: |
Gasoline |
Propane |
Kerosene |
Acetylene |
Electrical storms |
Natural gas |
Overexposure to sun |
|
|
|
Box 8.1. Burn Severity Factors
1.Extent of body surface area burned
2.Depth of tissue damage
3.Age of person
4.Part of body burned
5.Past medical history
•If the burned areas are scattered, small and irregularly shaped, the rule of palm can be used. The palm of the burned person’s hand represents 1 % body surface area.
•If 10 % or more of the body surface of a child or 15 % or more of that of an adult is burned, the injury is considered serious. The person requires hospitalisation and fluid replacement to prevent shock.
2.Depth
•Two factors determine the depth of a burn wound: temperature of the burning agent and duration of exposure time.
•Previous terminology to describe burn depth was first, second and third degree. In recent years, these terms have been replaced by those more descriptive in nature: superficial partial-thickness, deep partial-thickness and fullthickness (Table 8.3).
•Superficial burns, such as those produced by sunburn, are not taken into consideration when assessing extent and depth.
|
|
|
|
|
|
114 |
Table 8.3 Classification of burn injury depth |
|
|
|
|
|
|
|
|
|
|
|
|
|
Degree of burn |
Cause of injury |
Depth of injury |
Appearance |
Treatment |
|
|
First degree |
Superficial sunburn |
Superficial damage to |
Erythematous, blanching on |
Complete healing |
|
|
|
|
epithelium |
pressure, no blisters |
within 3–5 days with |
|
|
|
Brief exposure to hot |
Tactile and pain sensations |
|
no scarring |
|
|
|
liquids or heat flash |
intact |
|
|
|
|
|
|
|
|
|
|
|
Superficial partial-thickness |
Brief exposure to flame, |
Destruction of epidermis, |
Moist, weepy, blanching on |
Complete healing |
|
|
(second degree) |
flash or hot liquids |
superficial damage to upper |
pressure, blisters, pink or red |
within 14–21 days with |
|
|
|
|
layer of dermis, epidermal |
colour |
no scarring |
|
|
|
|
appendages intact |
|
|
|
|
|
|
|
|
|
|
|
Deep partial-thickness (deep |
Exposure to flame, scalding |
Destruction of epidermis, |
Pale and less moist, no blanching |
Prolonged healing time |
|
|
second degree) |
liquids or hot tar |
damage to dermis, some |
or prolonged, deep pressure |
usually >21 days with |
|
|
|
|
epidermal appendages intact |
sensation intact, pinprick |
scarring. Skin grafting |
|
|
|
|
|
sensation absent |
may be necessary for |
|
|
|
|
|
|
improved functional |
|
|
|
|
|
|
and aesthetic outcome |
|
|
|
|
|
|
|
|
|
Full-thickness (third degree) |
Prolonged contact with flame, |
Complete destruction of |
Dry, leathery, pale, mottled |
Requires skin grafting |
|
|
|
steam, scalding liquids, hot |
epidermis, dermis and |
brown or red in colour; visible |
|
|
|
|
objects, chemicals or |
epidermal appendages; injury |
thrombosed vessels insensitive |
|
|
|
|
electrical current |
through most of the dermis |
to pain and pressure |
|
|
|
|
|
|
|
|
|
|
Full-thickness (fourth degree) |
Major electrical current, |
Complete destruction of |
Dry, black, mottled brown, white |
Requires skin grafting |
|
|
|
prolonged contact with heat |
epidermis, dermis and |
or red; no sensation and limited |
and likely amputation |
|
|
|
source (i.e. unconscious patient) |
epidermal appendages; injury |
movement of burned limbs or |
|
|
|
|
|
involving connective tissue, |
digits |
|
|
|
|
|
muscle and bone |
|
|
|
.J |
|
|
|
|
|
|
|
|
|
|
|
|
|
Knighton |
8 Nursing Management of the Burn-Injured Person |
115 |
|
|
•The skin is divided into three layers, which include the epidermis, dermis and subcutaneous tissue (Fig. 8.1).
3.Age
•For patients less than 2 years of age and greater than 50, there is a higher incidence of morbidity and mortality.
•Sadly, the infant, toddler and elderly are at increased risk for abuse by burning.
4.Part of the body burned
•Patients with burns to the face, neck, hands, feet or perineum have greater challenges to overcome and require the specialised care offered by a burn centre.
5.Past medical history
•Pre-existing cardiovascular, pulmonary or renal disease will be exacerbated by the burn injury.
•Persons with diabetes or peripheral vascular disease have a more difficult time with wound healing, especially on the legs and feet.
Hair follicle
Degree of burn
Superficial partial
thickness
(1st degree)
Deep partial thickness
(2nd degree)
Full thickness
(3rd and 4th degree)
Sweat gland
Structure
Epidermis
Dermis
Fat
Muscle
Bone
Fig. 8.1 Anatomy of burn tissue depth