- •Contents
- •Contributors and consultants
- •Not another boring foreword
- •A look at cardiac anatomy
- •A look at cardiac physiology
- •A look at ECG recordings
- •All about leads
- •Observing the cardiac rhythm
- •Monitor problems
- •A look at an ECG complex
- •8-step method
- •Recognizing normal sinus rhythm
- •A look at sinus node arrhythmias
- •Sinus arrhythmia
- •Sinus bradycardia
- •Sinus tachycardia
- •Sinus arrest
- •Sick sinus syndrome
- •A look at atrial arrhythmias
- •Premature atrial contractions
- •Atrial tachycardia
- •Atrial flutter
- •Atrial fibrillation
- •Wandering pacemaker
- •A look at junctional arrhythmias
- •Premature junctional contraction
- •Junctional escape rhythm
- •Accelerated junctional rhythm
- •Junctional tachycardia
- •A look at ventricular arrhythmias
- •Premature ventricular contraction
- •Idioventricular rhythms
- •Ventricular tachycardia
- •Ventricular fibrillation
- •Asystole
- •A look at AV block
- •First-degree AV block
- •Type I second-degree AV block
- •Type II second-degree AV block
- •Third-degree AV block
- •A look at pacemakers
- •Working with pacemakers
- •Evaluating pacemakers
- •A look at biventricular pacemakers
- •A look at radiofrequency ablation
- •A look at ICDs
- •A look at antiarrhythmics
- •Antiarrhythmics by class
- •Teaching about antiarrhythmics
- •A look at the 12-lead ECG
- •Signal-averaged ECG
- •A look at 12-lead ECG interpretation
- •Disorders affecting a 12-lead ECG
- •Identifying types of MI
- •Appendices and index
- •Practice makes perfect
- •ACLS algorithms
- •Brushing up on interpretation skills
- •Look-alike ECG challenge
- •Quick guide to arrhythmias
- •Glossary
- •Selected references
- •Index
- •Notes
8
Atrioventricular blocks
Just the facts
In this chapter, you’ll learn:
the proper way to identify the various forms of atrioventricular (AV) block and interpret their rhythms
the reason that AV block is a significant arrhythmia
patients who at risk for developing AV block
signs and symptoms of AV block
nursing care for patients with AV block.
A look at AV block
Atrioventricular (AV) heart block results from an interruption in the conduction of impulses between the atria and ventricles. AV block can be total or partial or it may delay conduction. The block can occur at the AV node, the bundle of His, or the bundle branches.
The heart’s electrical impulses normally originate in the sinoatrial (SA) node, so when those impulses are blocked at the AV node, atrial rates are commonly normal (60 to 100 beats/minute). The clinical effect of the block depends on how many impulses are completely blocked, how slow the ventricular rate is as a result, and how the block ultimately affects the heart. A slow ventricular rate can decrease cardiac output, possibly causing lightheadedness, hypotension, and confusion.
The cause before the block
Various factors can lead to AV block, including underlying heart conditions, use of certain drugs, congenital anomalies, and conditions that disrupt the cardiac conduction system. (See Causes of AV block, page 154.)
In AV block, the impulses between the atria and ventricles are totally stopped, partially stopped, or delayed.
ATRIOVENTRICULAR BLOCKS
154
Causes of AV block
Atrioventricular (AV) blocks can be temporary or permanent. Here’s a look at causes of each kind of AV block.
Causes of temporary block
•Myocardial infarction (MI), usually inferior wall MI
•Digoxin (Lanoxin) toxicity
•Acute myocarditis
•Calcium channel blockers
•Beta-adrenergic blockers
•Cardiac surgery
Causes of permanent block
•Changes associated with aging
•Congenital abnormalities
•MI, usually anteroseptal MI
•Cardiomyopathy
•Cardiac surgery
Typical examples include:
•myocardial ischemia, which impairs cellular function so cells repolarize more slowly or incompletely. The injured cells, in turn, may conduct impulses slowly or inconsistently. Relief of the ischemia can restore normal function to the AV node.
•myocardial infarction (MI), in which cell death occurs. If the necrotic cells are part of the conduction system, they no longer conduct impulses and a permanent AV block occurs.
•excessive dosage of, or an exaggerated response to, a drug which can cause AV block or increase the likelihood that a block will develop. Although many antiarrhythmic medications can have this effect, the drugs more commonly known to cause or exacerbate AV blocks include digoxin, beta-adrenergic blockers, and calcium channel blockers.
•congenital anomalies such as congenital ventricular septal defect that involve cardiac structures and affect the conduction system. Anomalies of the conduction system, such as an AV node that doesn’t conduct impulses, can also occur in the absence of structural defects. (See AV block in elderly patients.)
Under the knife
AV block may also be caused by inadvertent damage to the heart’s conduction system during cardiac surgery. Damage is most likely to occur in surgery involving the mitral or tricuspid valve or in the closure of a ventricular septal defect. If the injury involves tissues
Ages
and stages
AV block in elderly patients
In elderly patients, atrioventricular (AV) block may be due to fibrosis of the conduction system. Other causes include use of digoxin and the presence of aortic valve calcification.
FIRST-DEGREE AV BLOCK |
155 |
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adjacent to the surgical site and the conduction system isn’t physically disrupted, the block may be only temporary. If a portion of the conduction system itself is severed, a permanent block results.
Radio blackout
Similar disruption of the conduction system can occur from a procedure called radiofrequency ablation. In this invasive procedure, a transvenous catheter is used to locate the area within the heart that participates in initiating or perpetuating certain tachyarrhythmias.
Radiofrequency energy is then delivered to the myocardium through this catheter to produce a small area of necrosis. The damaged tissue can no longer cause or participate in the tachyarrhythmia. If the energy is delivered close to the AV node, bundle of His, or bundle branches, block can occur.
Degrees of block
AV blocks are classified according to their severity, not their location. That severity is measured according to how well the node conducts impulses and is separated by degrees—first, second, and third. Let’s take a look at them one at a time.
First-degree AV block
First-degree AV block occurs when impulses from the atria are consistently delayed during conduction through the AV node. Conduction eventually
it just takes longer than normal. It’s as if people are walking in a line through a doorway, but each person hesitates before crossing the threshold.
How it happens
First-degree AV block may appear normally in a healthy person or result from myocardial
ischemia or infarction, myocarditis, or degenera-
tive changes in the heart. The condition may also be caused by medications, such as digoxin, calcium channel blockers, and betaadrenergic blockers.
First-degree AV block may be temporary, particularly if it stems from medications or ischemia early in the course of an MI. The presence of first-degree block, the least dangerous type of AV block, indicates some kind of problem in the conduction system.
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Identifying first-degree AV block
This rhythm strip illustrates first-degree atrioventricular (AV) block. Look for these distinguishing characteristics.
The PR interval is greater than 0.20 second.
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The rhythm is regular. |
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• Rhythm: Regular |
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• PR interval: 0.32 second |
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• QT interval: 0.40 second |
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• Rate: 75 beats/minute |
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• QRS complex: 0.08 second |
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• Other: PR interval prolonged but |
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• P wave: Normal |
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• T wave: Normal |
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constant |
Because first-degree AV block can progress to a more severe block, it should be monitored for changes.
What to look for
In general, a rhythm strip with this block looks like normal sinus rhythm except that the PR interval is longer than normal. (See
Identifying first-degree AV block.) The rhythm will be regular, with one normal P wave for every QRS complex.
The PR interval will be greater than 0.20 second and will be consistent for each beat. The QRS complex is usually normal, although sometimes a bundle-branch block may occur along with first-degree AV block and cause a widening of the QRS complex.
No signs of block
Most patients with first-degree AV block show no symptoms of the block because cardiac output isn’t significantly affected. If the PR interval is extremely long, a longer interval between S1 and S2 may be noted on cardiac auscultation.