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Chapter 22.  Approach to Abdominal Masses 217

FIGURE 22.7.  Sixteen-year-old female with (a) a large abdominal mass, (b) with a heterogenous appearance on CT scan, (c) resected via a laparotomy with a mixed solid and cystic appearance which was determined to be an ovarian teratoma on pathology.

Suggested Readings

1.Golden CB, Feusner JH. Malignant abdominal masses in children: quick guide to evaluation and diagnosis. Pediatr Clin North Am. 2002;49(6):1369-1392.

2.Seidel FG. Imaging of a pediatric abdominal mass. Semin Surg Oncol. 1986;2(3):125-138.

3.Kaste SC, McCarville MB. Imaging pediatric abdominal tumors. Semin Roentgenol. 2008;43(1):50-59.

4.Caty MG, Shamberger RC. Abdominal tumors in infancy and childhood. Pediatr Clin North Am. 1993;40(6):1253-1271.

5.Pearl RH, Irish MS, Caty MG, Glick PL. The approach to common abdominal diagnoses in infants and children. Part II. Pediatr Clin North Am. 1998;45(6):1287-1326.

Chapter 23

Gastro-Esophageal Reflux

Disease

David I. Campbell

Key Points

››Gastro-esophageal reflux disease (GORD) may present in a variety of ways and the presentation may vary according to age.

››Simple GORD may be treated empirically depending on age of the child.

››Troublesome GORD will require investigations such as endoscopy and pH run or a contrast study.

››Severe GORD is oftern associated with cow’s milk protein intolerance in children.

23.1  Introduction

Consider infants and young pre-school children as different from older children. The vomiting child is very obvious, but the consequences of reflux esophagitis can be marked with a lesser grade of reflux (GORD without vomiting) (Tables 23.1 and 23.2).

P.P. Godbole et al. (eds.), Guide to Pediatric Urology and

219

Surgery in Clinical Practice, DOI: 10.1007/978-1-84996-366-4_23,

© Springer-Verlag London Limited 2011

220 D.I. Campbell

TABLE 23.1.  Consider differential diagnoses in suspected GORD with the following components of GORD. Presentation and differential diagnosis of GORD in young children.

Profuse vomiting

3 months pyloric stenosis

 

(dry FTT, inexorable history).

 

UTI

 

Raised intracranial pressure

 

Metabolic diseases

Failure to Thrive

Due to calorie loss (i.e., vomiting,

 

see above)

 

Due to insufficient calorie intake

 

(i.e., feed aversion (see below)).

 

General medical clerking to identify

 

other body systems involved.

 

Always consider Cystic Fibrosis,

 

coeliac in the weaned child and

 

neglect.

Feed aversion

GORD may be co-existent with

 

milk allergy

 

Any cause of feed induced pain

 

(peptic ulcer, small bowel stricture

 

or ulcer, esophagitis)

Feeding problems

Neurological problems such as

(Gagging or choking)

CP/bulbar or pseudobulbar palsies

Hematemesis

Esophageal varices

 

Peptic ulcer

 

Swallowed blood (pharyngitis

 

or cracked nipples in breast fed

 

babies)

Poor sleep

Raised intracranial pressure

Sandifer Syndrome

Dystonia syndromes, epilepsy

 

(rare situations)

Drooling

Causes of hypotonia

Colic and abdominal pain

UTI, constipation, milk allergy

 

(often co-exists with GORD)

Chest infections

Cystic fibrosis

 

Primary immunodeficiency